(Q53312031)

13 May 2018

title

Complement activation in amyloid plaques in Alzheimer's disease brains does not proceed further than C3. (English)

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13 May 2018

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R Veerhuis

1

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13 May 2018

P van der Valk

2

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13 May 2018

I Janssen

3

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13 May 2018

S S Zhan

4

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13 May 2018

W E Van Nostrand

5

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13 May 2018

P Eikelenboom

6

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13 May 2018

publication date

1 January 1995

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13 May 2018

published in

Virchows Archiv

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13 May 2018

volume

426

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13 May 2018

issue

6

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13 May 2018

page(s)

603-610

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13 May 2018

https://scigraph.springernature.com/pub.10.1007/bf00192116

exact match

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cites work

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Immunohistochemical determination of complement activation in joint tissues of patients with rheumatoid arthritis and osteoarthritis using neoantigen-specific monoclonal antibodies

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beta-Amyloid activates complement by binding to a specific region of the collagen-like domain of the C1q A chain.

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A Monoclonal Antibody That Reacts Immunohistochemically with Amyloid Deposits in the Brain Tissue of Alzheimer Patients Binds to an Epitope Present on Complement Factor 4

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Immunoglobulins and complement factors in senile plaques. An immunoperoxidase study.

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Complement gene expression in neuroblastoma and astrocytoma cell lines of human origin

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12 December 2020

Protease nexin I immunostaining in Alzheimer's disease.

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12 December 2020

Monoclonal antibodies recognizing a neoantigen of poly(C9) detect the human terminal complement complex in tissue and plasma

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12 December 2020

The attachment of serum- and plasma-derived C3 to solid-phase immune aggregates and its relation to complement-mediated solubilization of immune complexes

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12 December 2020

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12 December 2020

Cerebral amyloid plaques in Alzheimer's disease but not in scrapie-affected mice are closely associated with a local inflammatory process

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Ultrastructural localization of complement membrane attack complex (MAC)-like immunoreactivity in brains of patients with Alzheimer's disease

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Expression of the complement classical pathway by human glioma in culture. A model for complement expression by nerve cells.

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Inhibitor of C1q secretion suppresses the macrophage response to lipid A for nitric oxide but not for TNF production: evidence for a role of C1q in autocrine binding of TNF

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12 December 2020

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12 December 2020

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12 December 2020

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12 December 2020

Protease nexin-1, an antithrombin with neurite outgrowth activity, is reduced in Alzheimer disease

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12 December 2020

Identifiers

DOI

10.1007/BF00192116

1 reference

13 May 2018

1 reference

13 May 2018