(Q53390124)

13 May 2018

title

Microglial activation is required for Abeta clearance after intracranial injection of lipopolysaccharide in APP transgenic mice. (English)

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13 May 2018

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Donna L Herber

1

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13 May 2018

Mary Mercer

2

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Lisa M Roth

3

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13 May 2018

Keisha Symmonds

4

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Jessica Maloney

5

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Nedda Wilson

6

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Melissa J Freeman

7

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Dave Morgan

8

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Marcia N Gordon

9

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13 May 2018

publication date

27 March 2007

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13 May 2018

Journal of Neuroimmune Pharmacology

published in

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13 May 2018

volume

2

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13 May 2018

issue

2

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13 May 2018

page(s)

222-231

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13 May 2018

https://scigraph.springernature.com/pub.10.1007/s11481-007-9069-z

exact match

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cites work

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Ibuprofen suppresses plaque pathology and inflammation in a mouse model for Alzheimer's disease.

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Intracranially Administered Anti-Αβ Antibodies Reduce β-Amyloid Deposition by Mechanisms Both Independent of and Associated with Microglial Activation

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Accelerated Alzheimer-type phenotype in transgenic mice carrying both mutant amyloid precursor protein and presenilin 1 transgenes

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Lipopolysaccharide-induced-neuroinflammation increases intracellular accumulation of amyloid precursor protein and amyloid beta peptide in APPswe transgenic mice

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Glucocorticoid- and mineralocorticoid receptors in microglial cells: the two receptors mediate differential effects of corticosteroids.

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Inflammation and primary demyelination induced by the intraspinal injection of lipopolysaccharide.

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Dynamic complexity of the microglial activation response in transgenic models of amyloid deposition: implications for Alzheimer therapeutics

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Complement component C1q modulates the phagocytosis of Abeta by microglia

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Endogenous brain cytokine mRNA and inflammatory responses to lipopolysaccharide are elevated in the Tg2576 transgenic mouse model of Alzheimer's disease

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Bone-marrow-derived cells contribute to the recruitment of microglial cells in response to beta-amyloid deposition in APP/PS1 double transgenic Alzheimer mice.

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Glucocorticoids increase amyloid-beta and tau pathology in a mouse model of Alzheimer's disease.

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Inflammation and cerebral amyloidosis are disconnected in an animal model of Alzheimer's disease

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Exaggerated astrocyte reactivity after nigrostriatal deafferentation in the aged rat.

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Inflammation and Alzheimer's disease

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Signal transduction by the lipopolysaccharide receptor, Toll-like receptor-4

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Regulation of pinocytosis in murine macrophages by colony-stimulating factors and other agents

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Glucocorticoids play a fundamental role in protecting the brain during innate immune response.

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Dexamethasone inhibits the antigen presentation of dendritic cells in MHC class II pathway

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12 December 2020

Microglial activation facilitates Abeta plaque removal following intracranial anti-Abeta antibody administration

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Effects of rofecoxib or naproxen vs placebo on Alzheimer disease progression: a randomized controlled trial

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Dexamethasone selectively suppresses microglial trophic responses to hippocampal deafferentation

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Microglial activation and beta -amyloid deposit reduction caused by a nitric oxide-releasing nonsteroidal anti-inflammatory drug in amyloid precursor protein plus presenilin-1 transgenic mice.

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Microglial phagocytosis of fibrillar beta-amyloid through a beta1 integrin-dependent mechanism.

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Immunization with amyloid-beta attenuates Alzheimer-disease-like pathology in the PDAPP mouse

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Diverse microglial responses after intrahippocampal administration of lipopolysaccharide

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12 December 2020

Intrahippocampal LPS injections reduce Abeta load in APP+PS1 transgenic mice

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12 December 2020

Time-dependent reduction in Abeta levels after intracranial LPS administration in APP transgenic mice.

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12 December 2020

Neuroinflammation-induced acceleration of amyloid deposition in the APPV717F transgenic mouse

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Scavenger receptors in neurobiology and neuropathology: their role on microglia and other cells of the nervous system

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IL-10 and glucocorticoids inhibit Aβ(1–42)- and lipopolysaccharide-induced pro-inflammatory cytokine and chemokine induction in the central nervous system

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12 December 2020

10.1007/S11481-007-9069-Z

Identifiers

DOI

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13 May 2018

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13 May 2018