(Q53391777)

13 May 2018

title

TAGing APP constrains neurogenesis. (English)

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13 May 2018

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2

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13 May 2018

author name string

Mark P Mattson

series ordinal

1

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13 May 2018

publication date

1 March 2008

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13 May 2018

published in

Nature Cell Biology

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13 May 2018

volume

10

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13 May 2018

issue

3

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13 May 2018

page(s)

249-250

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13 May 2018

https://scigraph.springernature.com/pub.10.1038/ncb0308-249

exact match

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cites work

The intracellular domain of the beta-amyloid precursor protein is stabilized by Fe65 and translocates to the nucleus in a notch-like manner

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The beta-amyloid precursor protein and its derivatives: from biology to learning and memory processes

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For the long run: maintaining germinal niches in the adult brain

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Notch and Presenilin: regulated intramembrane proteolysis links development and degeneration.

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A TAG1-APP signalling pathway through Fe65 negatively modulates neurogenesis

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Pathways towards and away from Alzheimer's disease

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Glycosylphosphatidylinositol anchored recognition molecules that function in axonal fasciculation, growth and guidance in the nervous system

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Notch targets the Cdk inhibitor Xic1 to regulate differentiation but not the cell cycle in neurons

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A heparin-binding domain in the amyloid protein precursor of Alzheimer's disease is involved in the regulation of neurite outgrowth

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Soluble form of amyloid precursor protein regulates proliferation of progenitors in the adult subventricular zone.

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Disruption of neurogenesis by amyloid beta-peptide, and perturbed neural progenitor cell homeostasis, in models of Alzheimer's disease.

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Expression of the axon growth-related neural adhesion molecule TAG-1/axonin-1 in the adult mouse brain.

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Exercise enhances learning and hippocampal neurogenesis in aged mice.

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Identifiers

DOI

10.1038/NCB0308-249

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13 May 2018

PubMed ID

18311177

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13 May 2018