(Q53406447)

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Chromosomal instability and p53 inactivation are required for genesis of glioblastoma but not for colorectal cancer in patients with germline mismatch repair gene mutation.

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10962567%20AND%20SRC:MED&resulttype=core&format=json

9 November 2019

Chromosomal instability and p53 inactivation are required for genesis of glioblastoma but not for colorectal cancer in patients with germline mismatch repair gene mutation (English)

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10962567%20AND%20SRC:MED&resulttype=core&format=json

9 November 2019

colorectal cancer

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1 reference

based on heuristic

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DNA mismatch repair

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1

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9 November 2019

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Yuen ST

2

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9 November 2019

Chan TL

3

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9 November 2019

Chan AS

4

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9 November 2019

Ho JW

5

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9 November 2019

Kwan K

6

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9 November 2019

Fan YW

7

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9 November 2019

Hung KN

8

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9 November 2019

Chung LP

9

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9 November 2019

Wyllie AH

10

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9 November 2019

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1 August 2000

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9 November 2019

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9 November 2019

19

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9 November 2019

35

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9 November 2019

4079-4083

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9 November 2019

https://scigraph.springernature.com/pub.10.1038/sj.onc.1203740

0 references

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Pathways leading to glioblastoma multiforme: a molecular analysis of genetic alterations in 65 astrocytic tumors.

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Microsatellite instability and mutation of DNA mismatch repair genes in gliomas.

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The TP53 tumour suppressor gene in colorectal carcinomas. II. Relation to DNA ploidy pattern and clinicopathological variables

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Drastic genetic instability of tumors and normal tissues in Turcot syndrome.

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Detection of multiple gains and losses of genetic material in ten glioma cell lines by comparative genomic hybridization.

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Investigation of genetic alterations associated with the grade of astrocytic tumor by comparative genomic hybridization

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Alternative genetic pathways in colorectal carcinogenesis.

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PTEN (MMAC1) mutations are frequent in primary glioblastomas (de novo) but not in secondary glioblastomas

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Molecular pathways in the formation of gliomas.

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Subsets of glioblastoma multiforme defined by molecular genetic analysis.

1 reference

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21 January 2018

Overexpression of the EGF receptor and p53 mutations are mutually exclusive in the evolution of primary and secondary glioblastomas.

1 reference

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Wild-type p53 restores cell cycle control and inhibits gene amplification in cells with mutant p53 alleles.

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Colorectal carcinoma in Hong Kong: epidemiology and genetic mutations.

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Genomic instability in colorectal carcinomas: comparison of different evaluation methods and their biological significance

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Deficiency of p53 accelerates mammary tumorigenesis in Wnt-1 transgenic mice and promotes chromosomal instability

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Chromosome number and structure both are markedly stable in RER colorectal cancers and are not destabilized by mutation of p53

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Microsatellite instability in sporadic carcinomas of the proximal colon: association with diploid DNA content, negative protein expression of p53, and distinct histomorphologic features

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Genomic instability and apoptosis are frequent in p53 deficient young mice

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Reliability of differential PCR for the detection of EGFR and MDM2 gene amplification in DNA extracted from FFPE glioma tissue.

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Altered cell cycle arrest and gene amplification potential accompany loss of wild-type p53

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7 January 2021

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DNA-repeat instability is associated with colorectal cancers presenting minimal chromosome rearrangements

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Detection of complex genetic alterations in human glioblastoma multiforme using comparative genomic hybridization.

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7 January 2021

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inferred from DOI database lookup

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