(Q53440352)

14 May 2018

title

p53 loss and point mutations are associated with suppression of apoptosis and progression of CML into myeloid blastic crisis. (English)

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14 May 2018

author

Liborio Stuppia

1

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14 May 2018

author name string

Calabrese G

2

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14 May 2018

Peila R

3

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14 May 2018

Guanciali-Franchi P

4

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14 May 2018

Morizio E

5

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14 May 2018

Spadano A

6

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14 May 2018

Palka G

7

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14 May 2018

publication date

1 October 1997

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14 May 2018

Cancer Genetics and Cytogenetics

published in

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14 May 2018

volume

98

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14 May 2018

issue

1

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14 May 2018

page(s)

28-35

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14 May 2018

Philadelphia chromosomal breakpoints are clustered within a limited region, bcr, on chromosome 22

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Hemizygous expression of the wild-type p53 allele may confer a selective growth advantage before complete inactivation of the p53 gene in the progression of chronic myelogenous leukaemia

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p53-dependent apoptosis suppresses tumor growth and progression in vivo

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Wild-type p53 induces apoptosis of myeloid leukaemic cells that is inhibited by interleukin-6

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Expression of different conformations of p53 in the blast cells of acute myeloblastic leukaemia is related to in vitro growth characteristics

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Simultaneous determination of cell surface antigens and apoptosis

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Comparison of methods for the detection of in situ restriction enzyme – nick translation using fluorochromes and confocal microscopy

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Prognostic significance of loss of a chromosome 17p and p53 gene mutations in blast crisis of chronic myelogenous leukaemia

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Identifiers

DOI

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14 May 2018

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14 May 2018