(Q53518091)

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Palmitate-induced inflammatory pathways in human adipose microvascular endothelial cells promote monocyte adhesion and impair insulin transcytosis.

scientific article published on 5 May 2015

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Europe PubMed Central

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7 March 2020

Palmitate-induced inflammatory pathways in human adipose microvascular endothelial cells promote monocyte adhesion and impair insulin transcytosis (English)

1 reference

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7 March 2020

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Nicolas J. Pillon

1

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7 March 2020

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7 March 2020

Kathryn E Hopperton

7

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7 March 2020

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7 March 2020

author name string

Paymon M Azizi

2

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7 March 2020

Yujin E Li

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7 March 2020

Jun Liu

4

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7 March 2020

Changsen Wang

5

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7 March 2020

Kenny L Chan

6

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7 March 2020

Richard P Bazinet

8

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7 March 2020

Philip J Bilan

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7 March 2020

Amira Klip

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7 March 2020

publication date

5 May 2015

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7 March 2020

American Journal of Physiology - Endocrinology and Metabolism

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7 March 2020

309

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7 March 2020

1

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7 March 2020

E35-44

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7 March 2020

Endothelial cell heterogeneity

1 reference

https://api.crossref.org/works/10.1152%2FAJPENDO.00611.2014

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Mitochondrial and cellular mechanisms for managing lipid excess

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Clathrin-dependent entry and vesicle-mediated exocytosis define insulin transcytosis across microvascular endothelial cells

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Exocytotic vesicle behaviour assessed by total internal reflection fluorescence microscopy

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AMPK inhibits fatty acid-induced increases in NF-kappaB transactivation in cultured human umbilical vein endothelial cells.

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p38 mitogen-activated protein kinase mediates palmitate-induced apoptosis but not inhibitor of nuclear factor-kappaB degradation in human coronary artery endothelial cells

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Direct administration of insulin into skeletal muscle reveals that the transport of insulin across the capillary endothelium limits the time course of insulin to activate glucose disposal

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Adiponectin inhibits palmitate-induced apoptosis through suppression of reactive oxygen species in endothelial cells: involvement of cAMP/protein kinase A and AMP-activated protein kinase

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Role of lipotoxicity in endothelial dysfunction

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Diet-induced obesity prevents interstitial dispersion of insulin in skeletal muscle

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Sulfo-N-succinimidyl oleate (SSO) inhibits fatty acid uptake and signaling for intracellular calcium via binding CD36 lysine 164: SSO also inhibits oxidized low density lipoprotein uptake by macrophages

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21 January 2018

Eicosapentaenoic acid protects against palmitic acid-induced endothelial dysfunction via activation of the AMPK/eNOS pathway

1 reference

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21 January 2018

Activation of NF-kappaB by palmitate in endothelial cells: a key role for NADPH oxidase-derived superoxide in response to TLR4 activation

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21 January 2018

Hematopoietic cell-restricted deletion of CD36 reduces high-fat diet-induced macrophage infiltration and improves insulin signaling in adipose tissue

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Induction of stearoyl-CoA desaturase protects human arterial endothelial cells against lipotoxicity

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Leukocyte migration in adipose tissue of mice null for ICAM-1 and Mac-1 adhesion receptors

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Fasting nonesterified fatty acid profiles in childhood and their relationship with adiposity, insulin sensitivity, and lipid levels

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On, around, and through: neutrophil-endothelial interactions in innate immunity

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TLR4 links innate immunity and fatty acid-induced insulin resistance

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Unraveling the cellular mechanism of insulin resistance in humans: new insights from magnetic resonance spectroscopy

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Protein kinase C iota mediates lipid-induced apoptosis of human coronary artery endothelial cells

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Circulating palmitoleate strongly and independently predicts insulin sensitivity in humans

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Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance

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Berberine protects against palmitate-induced endothelial dysfunction: involvements of upregulation of AMPK and eNOS and downregulation of NOX4.

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