(Q53651197)

16 May 2018

title

Tau and GSK3beta dephosphorylations are required for regulating Pin1 phosphorylation. (English)

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16 May 2018

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Sae H Min

1

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16 May 2018

Jung S Cho

2

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16 May 2018

Jae H Oh

3

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16 May 2018

Sun B Shim

4

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16 May 2018

Dae Y Hwang

5

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16 May 2018

Su H Lee

6

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16 May 2018

Seung W Jee

7

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16 May 2018

Hwa J Lim

8

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16 May 2018

Min Y Kim

9

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16 May 2018

Yhun Y Sheen

10

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16 May 2018

Seok H Lee

11

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16 May 2018

Yong K Kim

12

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16 May 2018

publication date

1 August 2005

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16 May 2018

published in

Neurochemical Research

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16 May 2018

volume

30

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16 May 2018

issue

8

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16 May 2018

page(s)

955-961

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16 May 2018

https://scigraph.springernature.com/pub.10.1007/s11064-005-6177-0

exact match

0 references

cites work

The prolyl isomerase Pin1 restores the function of Alzheimer-associated phosphorylated tau protein

1 reference

12 December 2020

GSK-3alpha regulates production of Alzheimer's disease amyloid-beta peptides

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12 December 2020

Glycogen synthase kinase-3 induces Alzheimer's disease-like phosphorylation of tau: generation of paired helical filament epitopes and neuronal localisation of the kinase.

1 reference

12 December 2020

Inactivation of Integrin-linked Kinase Induces Aberrant Tau Phosphorylation via Sustained Activation of Glycogen Synthase Kinase 3β in N1E-115 Neuroblastoma Cells

1 reference

12 December 2020

Transgenic mice expressing beta-galactosidase in mature neurons under neuron-specific enolase promoter control

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12 December 2020

The peptidyl prolyl cis/trans-isomerase Pin1 recognizes the phospho-Thr212-Pro213 site on Tau.

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12 December 2020

Lithium reduces tau phosphorylation by inhibition of glycogen synthase kinase-3.

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12 December 2020

Alzheimer's disease-like phosphorylation of the microtubule-associated protein tau by glycogen synthase kinase-3 in transfected mammalian cells.

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12 December 2020

Requirement of the prolyl isomerase Pin1 for the replication checkpoint.

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12 December 2020

Aberrant expressions of pathogenic phenotype in Alzheimer's diseased transgenic mice carrying NSE-controlled APPsw.

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12 December 2020

A molecular mechanism for the effect of lithium on development

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12 December 2020

Lithium reduces tau phosphorylation: effects in living cells and in neurons at therapeutic concentrations.

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12 December 2020

Shortfalls in the peptidyl-prolyl cis-trans isomerase protein Pin1 in neurons are associated with frontotemporal dementias.

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12 December 2020

Pathways towards and away from Alzheimer's disease

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12 December 2020

Lithium inhibits neurite growth and tau protein kinase I/glycogen synthase kinase-3beta-dependent phosphorylation of juvenile tau in cultured hippocampal neurons.

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12 December 2020

Role of the prolyl isomerase Pin1 in protecting against age-dependent neurodegeneration.

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12 December 2020

Lithium inhibits Alzheimer's disease-like tau protein phosphorylation in neurons.

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12 December 2020

Glycogen synthase kinase 3β is identical to tau protein kinase I generating several epitopes of paired helical filaments

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12 December 2020

Lithium inhibits amyloid secretion in COS7 cells transfected with amyloid precursor protein C100.

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12 December 2020

Pin1 colocalization with phosphorylated tau in Alzheimer's disease and other tauopathies

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12 December 2020

A human peptidyl-prolyl isomerase essential for regulation of mitosis

1 reference

12 December 2020

10.1007/S11064-005-6177-0

Identifiers

DOI

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16 May 2018

PubMed ID

16258844

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16 May 2018