(Q53753971)

19 May 2018

title

Continuous treatment with a low-dose β-agonist reduces bone mass by increasing bone resorption without suppressing bone formation. (English)

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19 May 2018

author name string

Hisataka Kondo

series ordinal

1

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19 May 2018

Akifumi Togari

series ordinal

2

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19 May 2018

language of work or name

English

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publication date

30 September 2010

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19 May 2018

Calcified Tissue International

published in

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19 May 2018

volume

88

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19 May 2018

issue

1

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19 May 2018

page(s)

23-32

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19 May 2018

https://scigraph.springernature.com/pub.10.1007/s00223-010-9421-9

exact match

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cites work

Characterization of beta-adrenergic receptors on rat and human osteoblast-like cells and demonstration that beta-receptor agonists can stimulate bone resorption in organ culture

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Expression of mRNAs for neuropeptide receptors and beta-adrenergic receptors in human osteoblasts and human osteogenic sarcoma cells

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Leptin inhibits bone formation through a hypothalamic relay: a central control of bone mass

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Central control of bone formation

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Adrenergic stimulation of osteoclastogenesis mediated by expression of osteoclast differentiation factor in MC3T3-E1 osteoblast-like cells

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Adrenergic regulation of bone metabolism: possible involvement of sympathetic innervation of osteoblastic and osteoclastic cells

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Leptin regulates bone formation via the sympathetic nervous system

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In vivo stimulation of sympathetic nervous system modulates osteoblastic activity in mouse calvaria

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Effects of beta-adrenergic agonists on bone-resorbing activity in human osteoclast-like cells

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Serum leptin level is a regulator of bone mass

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Leptin regulation of bone resorption by the sympathetic nervous system and CART

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The role of the sympathetic nervous system in controlling bone metabolism

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Unloading induces osteoblastic cell suppression and osteoclastic cell activation to lead to bone loss via sympathetic nervous system

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Alteration of trabecular bone under chronic beta2 agonists treatment

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Severe bone alterations under beta2 agonist treatments: bone mass, microarchitecture and strength analyses in female rats

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Osteopontin Deficiency Induces Parathyroid Hormone Enhancement of Cortical Bone Formation

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Beta2-adrenoreceptor ligands regulate osteoclast differentiation in vitro by direct and indirect mechanisms

21 January 2018

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The effects of parathyroid hormone and alendronate alone or in combination in postmenopausal osteoporosis

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21 January 2018

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10.1007/S00223-010-9421-9

21 January 2018

Identifiers

DOI

1 reference

19 May 2018

1 reference

19 May 2018