(Q54032075)

23 May 2018

title

Chronic myocardial hypoxia increases nitric oxide synthase and decreases caveolin-3. (English)

1 reference

23 May 2018

0 references

Y Shi

1

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23 May 2018

K A Pritchard

2

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23 May 2018

P Holman

3

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23 May 2018

P Rafiee

4

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23 May 2018

O W Griffith

5

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23 May 2018

B Kalyanaraman

6

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23 May 2018

J E Baker

7

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23 May 2018

publication date

1 October 2000

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23 May 2018

Free Radical Biology and Medicine

published in

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23 May 2018

volume

29

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23 May 2018

issue

8

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23 May 2018

page(s)

695-703

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23 May 2018

Adaptation to chronic hypoxia confers tolerance to subsequent myocardial ischemia by increased nitric oxide production

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Prolonged in vivo hypoxia enhances nitric oxide synthase type I and type III gene expression in adult rat lung

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Endothelial nitric oxide synthase targeting to caveolae. Specific interactions with caveolin isoforms in cardiac myocytes and endothelial cells

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7 January 2021

Endothelial nitric oxide synthase is regulated by tyrosine phosphorylation and interacts with caveolin-1

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7 January 2021

Modulation of the endothelial nitric-oxide synthase-caveolin interaction in cardiac myocytes. Implications for the autonomic regulation of heart rate.

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Direct interaction of endothelial nitric-oxide synthase and caveolin-1 inhibits synthase activity

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7 January 2021

Preconditioning in immature rabbit hearts: role of KATP channels.

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Simultaneous determination of nitrate and nitrite in biological samples by multichannel flow injection analysis

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The mechanism of cardioprotection by S-nitrosoglutathione monoethyl ester in rat isolated heart during cardioplegic ischaemic arrest

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Increased tolerance of the chronically hypoxic immature heart to ischemia. Contribution of the KATP channel

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The late phase of ischemic preconditioning is abrogated by targeted disruption of the inducible NO synthase gene

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Ischemic preconditioning increases iNOS transcript levels in conscious rabbits via a nitric oxide-dependent mechanism

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Ischaemic preconditioning: present position and future directions.

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Inhibition of 3-hydroxy-3-methylglutaryl (HMG)-CoA reductase blocks hypoxia-mediated down-regulation of endothelial nitric oxide synthase

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Transcriptional and posttranscriptional regulation of endothelial nitric oxide synthase expression by hydrogen peroxide

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7 January 2021

Posttranscriptional regulation of endothelial nitric oxide synthase during cell growth

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7 January 2021

Dynamic activation of endothelial nitric oxide synthase by Hsp90.

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7 January 2021

Hypercholesterolemia decreases nitric oxide production by promoting the interaction of caveolin and endothelial nitric oxide synthase

1 reference

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7 January 2021

10.1016/S0891-5849(00)00364-6

Identifiers

DOI

1 reference

23 May 2018

1 reference

23 May 2018