(Q54057103)

24 May 2018

title

Enhancement of Th2 response in IL-6-deficient mice immunized with myelin oligodendrocyte glycoprotein. (English)

1 reference

24 May 2018

0 references

1 reference

Okuda Y

1

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24 May 2018

Sakoda S

2

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24 May 2018

Saeki Y

3

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24 May 2018

Kishimoto T

4

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24 May 2018

Yanagihara T

5

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24 May 2018

publication date

1 June 2000

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24 May 2018

Journal of Neuroimmunology

published in

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24 May 2018

volume

105

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24 May 2018

issue

2

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24 May 2018

page(s)

120-123

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24 May 2018

IL-6-deficient mice resist myelin oligodendrocyte glycoprotein-induced autoimmune encephalomyelitis

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7 January 2021

A T helper cell 2 (Th2) immune response against non-self antigens modifies the cytokine profile of autoimmune T cells and protects against experimental allergic encephalomyelitis

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7 January 2021

Tumor necrosis factor alpha and lymphotoxin alpha are not required for induction of acute experimental autoimmune encephalomyelitis.

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7 January 2021

Interleukin 12 is required for the T-lymphocyte-independent induction of interferon gamma by an intracellular parasite and induces resistance in T-cell-deficient hosts

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7 January 2021

Administration of neutralizing antibodies to interleukin-6 (IL-6) reduces experimental autoimmune encephalomyelitis and is associated with elevated levels of IL-6 bioactivity in central nervous system and circulation

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Impaired immune and acute-phase responses in interleukin-6-deficient mice.

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Critical points of tumor necrosis factor action in central nervous system autoimmune inflammation defined by gene targeting

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TNF is a potent anti-inflammatory cytokine in autoimmune-mediated demyelination.

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Interleukin-6 functions in autoimmune encephalomyelitis: a study in gene-targeted mice

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Interleukin 6 plays a key role in the development of antigen-induced arthritis

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Expression of the inducible isoform of nitric oxide synthase in the central nervous system of mice correlates with the severity of actively induced experimental allergic encephalomyelitis

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7 January 2021

The development of autoimmune encephalomyelitis provoked by myelin oligodendrocyte glycoprotein is associated with an upregulation of both proinflammatory and immunoregulatory cytokines in the central nervous system

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The pattern of cytokine gene expression in lymphoid organs and peripheral blood mononuclear cells of mice with experimental allergic encephalomyelitis

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7 January 2021

IL-6-deficient mice are resistant to the induction of experimental autoimmune encephalomyelitis provoked by myelin oligodendrocyte glycoprotein

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7 January 2021

IL-6 plays a crucial role in the induction phase of myelin oligodendrocyte glucoprotein 35-55 induced experimental autoimmune encephalomyelitis.

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7 January 2021

Critical Influences of the Cytokine Orchestration on the Outcome of Myelin Antigen‐Specific T‐Cell Autoimmunity in Experimental Autoimmune Encephalomyelitis and Multiple Sclerosis

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7 January 2021

Challenging cytokine redundancy: inflammatory cell movement and clinical course of experimental autoimmune encephalomyelitis are normal in lymphotoxin-deficient, but not tumor necrosis factor-deficient, mice

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Impaired neutrophil response and CD4+ T helper cell 1 development in interleukin 6-deficient mice infected with Candida albicans

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An interleukin (IL)-10/IL-12 immunoregulatory circuit controls susceptibility to autoimmune disease.

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A critical role for lymphotoxin in experimental allergic encephalomyelitis

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Encephalitogenic Th1 cells are inhibited by Th2 cells with related peptide specificity: relative roles of interleukin (IL)-4 and IL-10.

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7 January 2021

10.1016/S0165-5728(00)00192-2

Identifiers

DOI

1 reference

24 May 2018

1 reference

24 May 2018