(Q54290446)

27 May 2018

title

ICAM-1 and VCAM-1 mediate endotoxemic myocardial dysfunction independent of neutrophil accumulation. (English)

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27 May 2018

author

Anirban Banerjee

6

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27 May 2018

author name string

Christopher D Raeburn

1

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27 May 2018

Casey M Calkins

2

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27 May 2018

Michael A Zimmerman

3

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27 May 2018

Yong Song

4

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27 May 2018

Lihua Ao

5

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27 May 2018

Alden H Harken

7

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27 May 2018

Xianzhong Meng

8

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27 May 2018

publication date

1 August 2002

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27 May 2018

American Journal of Physiology - Regulatory, Integrative and Comparative Physiology

published in

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27 May 2018

volume

283

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27 May 2018

issue

2

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27 May 2018

page(s)

R477-86

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27 May 2018

Tumor necrosis factor-alpha and interleukin-1beta synergistically depress human myocardial function

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21 January 2018

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Differential effects of caspase inhibitors on endotoxin-induced myocardial dysfunction and heart apoptosis

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Tumor necrosis factor alpha and interleukin 1beta are responsible for in vitro myocardial cell depression induced by human septic shock serum

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Fibrinogen mediates leukocyte adhesion to vascular endothelium through an ICAM-1-dependent pathway

21 January 2018

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Lymphocyte migration through monolayers of endothelial cell lines involves VCAM-1 signaling via endothelial cell NADPH oxidase

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A circulating myocardial depressant substance in humans with septic shock. Septic shock patients with a reduced ejection fraction have a circulating factor that depresses in vitro myocardial cell performance

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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Sphingosine Mediates the Immediate Negative Inotropic Effects of Tumor Necrosis Factor-α in the Adult Mammalian Cardiac Myocyte

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21 January 2018

Identifiers

DOI

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27 May 2018

PubMed ID

12121861

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27 May 2018