(Q54586672)

1 June 2018

title

Lipopolysaccharide and TNF-alpha produce very similar changes in gene expression in human endothelial cells. (English)

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1 June 2018

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4

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author name string

Magder S

series ordinal

1

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1 June 2018

Neculcea J

series ordinal

2

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Neculcea V

series ordinal

3

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publication date

17 August 2006

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1 June 2018

Journal of Vascular Research

published in

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1 June 2018

volume

43

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issue

5

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1 June 2018

page(s)

447-461

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Points of control in inflammation

cites work

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Innate immune recognition

21 January 2018

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Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

21 January 2018

1 reference

21 January 2018

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CD14-deficient mice are protected against lipopolysaccharide-induced cardiac inflammation and left ventricular dysfunction.

21 January 2018

1 reference

Lipopolysaccharide recognition: CD14, TLRs and the LPS-activation cluster

21 January 2018

1 reference

Oxidative stress in septic shock and disseminated intravascular coagulation

21 January 2018

1 reference

Chemokines in innate and adaptive host defense: basic chemokinese grammar for immune cells

21 January 2018

1 reference

Phosphorylation meets ubiquitination: the control of NF-[kappa]B activity

21 January 2018

1 reference

Modulation of toll-like receptor 4 expression on human monocytes by tumor necrosis factor and interleukin-6: tumor necrosis factor evokes lipopolysaccharide hyporesponsiveness, whereas interleukin-6 enhances lipopolysaccharide activity.

21 January 2018

1 reference

NAD(P)H oxidase mediates the endothelial barrier dysfunction induced by TNF-alpha

21 January 2018

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21 January 2018

Identifiers

DOI

10.1159/000095162

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1 June 2018

PubMed ID

16921252

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1 June 2018