(Q54651740)

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Attenuation of diabetic nephropathy in diabetes rats induced by streptozotocin by regulating the endoplasmic reticulum stress inflammatory response

scientific article published on September 3, 2010

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Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

Attenuation of diabetic nephropathy in diabetes rats induced by streptozotocin by regulating the endoplasmic reticulum stress inflammatory response (English)

1 reference

10.1016/J.METABOL.2010.07.021

https://api.crossref.org/works/10.1016/J.METABOL.2010.07.021

6 July 2024

endoplasmic reticulum

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diabetic nephropathy

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based on heuristic

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author name string

Wei Qi

1

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Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

Jiao Mu

2

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Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

Zhi-Feng Luo

3

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Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

Wei Zeng

4

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Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

Yan-Hong Guo

5

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Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

Qi Pang

6

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Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

Zi-Lin Ye

7

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Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

Li Liu

8

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Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

Fa-Huan Yuan

9

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Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

Bing Feng

10

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Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

language of work or name

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National Center for Biotechnology Information

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=20817186

6 July 2024

publication date

3 September 2010

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Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

full work available at URL

https://api.elsevier.com/content/article/PII:S0026049510002490?httpAccept=text/xml

1 reference

10.1016/J.METABOL.2010.07.021

https://api.crossref.org/works/10.1016/J.METABOL.2010.07.021

6 July 2024

https://api.elsevier.com/content/article/PII:S0026049510002490?httpAccept=text/plain

1 reference

10.1016/J.METABOL.2010.07.021

https://api.crossref.org/works/10.1016/J.METABOL.2010.07.021

6 July 2024

1 reference

Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

60

1 reference

Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

5

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Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

594-603

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Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

Lectin-like oxidized LDL receptor-1 (LOX-1) expression in the tubulointerstitial area likely plays an important role in human diabetic nephropathy.

1

1 reference

10.1016/J.METABOL.2010.07.021

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Inflammation and pathogenesis of diabetic nephropathy

2

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Role of inflammation in diabetic complications

3

1 reference

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Endoplasmic reticulum stress: another link between obesity and insulin resistance/inflammation?

4

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6 July 2024

Role of inflammation in diabetic nephropathy

5

1 reference

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From endoplasmic-reticulum stress to the inflammatory response

6

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Suppressive effects of 4-phenylbutyrate on the aggregation of Pael receptors and endoplasmic reticulum stress

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Sodium 4-phenylbutyrate protects against cerebral ischemic injury.

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Chemical chaperones reduce ER stress and restore glucose homeostasis in a mouse model of type 2 diabetes

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The preliminary studies on the cross action between endothelin-1 and angiotensin II in kidney tissue of rats with diabetic nephropathy

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High-fat diet in low-dose-streptozotocin-treated heminephrectomized rats induces all features of human type 2 diabetic nephropathy: a new rat model of diabetic nephropathy

11

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The rat model of type 2 diabetic mellitus and its glycometabolism characters

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A new rat model of type 2 diabetes: the fat-fed, streptozotocin-treated rat.

13

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Kidney involvement in a nongenetic rat model of type 2 diabetes

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Reactive oxygen species mediate high glucose-induced plasminogen activator inhibitor-1 up-regulation in mesangial cells and in diabetic kidney

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The breakdown of preexisting advanced glycation end products is associated with reduced renal fibrosis in experimental diabetes.

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Statistical analysis of real-time PCR data

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Orchestrating the unfolded protein response in health and disease

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The ER chaperone and signaling regulator GRP78/BiP as a monitor of endoplasmic reticulum stress

19

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Dynamic interaction of BiP and ER stress transducers in the unfolded-protein response

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Protein translation and folding are coupled by an endoplasmic-reticulum-resident kinase

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SP600125, a selective JNK inhibitor, protects ischemic renal injury via suppressing the extrinsic pathways of apoptosis

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Inflammation and endoplasmic reticulum stress in obesity and diabetes

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Oxidative stress, ER stress, and the JNK pathway in type 2 diabetes

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MCP-1/CCL2: a new diagnostic marker and therapeutic target for progressive renal injury in diabetic nephropathy

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Cisplatin, gentamicin, and p-aminophenol induce markers of endoplasmic reticulum stress in the rat kidneys.

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Dysfunction of the ER chaperone BiP accelerates the renal tubular injury

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Preconditioning with endoplasmic reticulum stress ameliorates mesangioproliferative glomerulonephritis

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Proteinuria and hyperglycemia induce endoplasmic reticulum stress

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Macrophage-mediated renal injury is dependent on signaling via the JNK pathway

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Possible relationship of monocyte chemoattractant protein-1 with diabetic nephropathy.

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Monocyte chemoattractant protein-1 promotes the development of diabetic renal injury in streptozotocin-treated mice

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Monocyte chemoattractant protein-1-induced tissue inflammation is critical for the development of renal injury but not type 2 diabetes in obese db/db mice.

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Elevated expression of transforming growth factor-beta and proteoglycan production in experimental glomerulonephritis. Possible role in expansion of the mesangial extracellular matrix

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Upregulation of type I collagen by TGF-beta in mesangial cells is blocked by PPARgamma activation

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Endoplasmic reticulum stress links obesity, insulin action, and type 2 diabetes

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Endoplasmic reticulum stress in proteinuric kidney disease

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The c-Jun NH(2)-terminal kinase promotes insulin resistance during association with insulin receptor substrate-1 and phosphorylation of Ser(307)

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Defective trafficking of nephrin missense mutants rescued by a chemical chaperone

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Identifiers

10.1016/J.METABOL.2010.07.021

1 reference

Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

2 June 2018

http://europepmc.org/abstract/MED/20817186

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