(Q54704228)

3 June 2018

title

Expression of inducible nitric oxide synthase is increased in patients with heart failure due to ischemic disease. (English)

1 reference

3 June 2018

0 references

C R Ferreiro

1

1 reference

3 June 2018

A C P Chagas

2

1 reference

3 June 2018

M H C Carvalho

3

1 reference

3 June 2018

A P Dantas

4

1 reference

3 June 2018

C Scavone

5

1 reference

3 June 2018

L C B Souza

6

1 reference

3 June 2018

E Buffolo

7

1 reference

3 June 2018

P L da Luz

8

1 reference

3 June 2018

language of work or name

English

0 references

publication date

24 August 2004

1 reference

3 June 2018

Brazilian Journal of Medical and Biological Research

published in

1 reference

3 June 2018

volume

37

1 reference

3 June 2018

issue

9

1 reference

3 June 2018

page(s)

1313-1320

1 reference

3 June 2018

The L-arginine-nitric oxide pathway

cites work

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

Molecular control of nitric oxide synthases in the cardiovascular system

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

Molecular biology and molecular genetics of nitric oxide synthase genes.

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

Nitric oxide synthase isozymes. Characterization, purification, molecular cloning, and functions.

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

Effects of damaging the endocardial surface on the mechanical performance of isolated cardiac muscle

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

Induction and potential biological relevance of a Ca(2+)-independent nitric oxide synthase in the myocardium

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

Myocardial calcium-independent nitric oxide synthase activity is present in dilated cardiomyopathy, myocarditis, and postpartum cardiomyopathy but not in ischaemic or valvar heart disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

Expression of inducible nitric oxide synthase in failing and non-failing human heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

Determination of left ventricular ejection fraction by computer densitometric analysis of digital subtraction angiography: experimental validation and correlation with area-length methods

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

Nitric oxide, cGMP, and hormone regulation of active sodium transport

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

Endothelial cell regulation of contractility of the heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

Nitric oxide synthases: which, where, how, and why?

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

Nitric oxide as a bifunctional regulator of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

Myocardial dysfunction in sepsis: mechanisms and therapeutic implications

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

Congenital deficiency of nitric oxide synthase 2 protects against endotoxin-induced myocardial dysfunction in mice.

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

Sphingosine Mediates the Immediate Negative Inotropic Effects of Tumor Necrosis Factor-α in the Adult Mammalian Cardiac Myocyte

21 January 2018

1 reference

https://api.crossref.org/works/10.1590%2FS0100-879X2004000900005

10.1590/S0100-879X2004000900005

21 January 2018

Identifiers

DOI

1 reference

3 June 2018

1 reference

3 June 2018