(Q54705484)

3 June 2018

title

The expression of interleukin-17, interferon-gamma, and macrophage inflammatory protein-3 alpha mRNA in patients with psoriasis vulgaris. (English)

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3 June 2018

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Jiawen Li

1

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3 June 2018

Dongsheng Li

series ordinal

2

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3 June 2018

Zhijian Tan

series ordinal

3

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3 June 2018

publication date

1 January 2004

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3 June 2018

Journal of Huazhong University of Science and Technology [Medical Sciences]

published in

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3 June 2018

volume

24

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3 June 2018

issue

3

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3 June 2018

page(s)

294-296

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3 June 2018

Up-regulation of macrophage inflammatory protein-3 alpha/CCL20 and CC chemokine receptor 6 in psoriasis.

cites work

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Crossref

https://api.crossref.org/works/10.1007%2FBF02832018

Overexpression of monocyte-derived cytokines in active psoriasis: a relation to coexistent arthropathy

21 January 2018

1 reference

12 December 2020

Interleukin-17 is produced by both Th1 and Th2 lymphocytes, and modulates interferon-gamma- and interleukin-4-induced activation of human keratinocytes

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12 December 2020

T cell interleukin-17 induces stromal cells to produce proinflammatory and hematopoietic cytokines

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12 December 2020

Interleukin-17 and interferon-gamma synergize in the enhancement of proinflammatory cytokine production by human keratinocytes.

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12 December 2020

Macrophage inflammatory protein 3alpha is expressed at inflamed epithelial surfaces and is the most potent chemokine known in attracting Langerhans cell precursors.

1 reference

12 December 2020

Enhancing effect of IL-1, IL-17, and TNF-alpha on macrophage inflammatory protein-3alpha production in rheumatoid arthritis: regulation by soluble receptors and Th2 cytokines

1 reference

12 December 2020

The majority of epidermal T cells in Psoriasis vulgaris lesions can produce type 1 cytokines, interferon-gamma, interleukin-2, and tumor necrosis factor-alpha, defining TC1 (cytotoxic T lymphocyte) and TH1 effector populations: a type 1 differentiat

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12 December 2020

Identifiers

DOI

10.1007/BF02832018

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3 June 2018

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3 June 2018