(Q55155932)

English

Sickle cell anemia, the first molecular disease: overview of molecular etiology, pathophysiology, and therapeutic approaches.

scientific article

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scholarly article

1 reference

Europe PubMed Central

PMC publication ID

23 June 2018

http://europepmc.org/abstract/PMC/5848659

1 reference

22 April 2019

Sickle cell anemia, the first molecular disease: overview of molecular etiology, pathophysiology, and therapeutic approaches. (English)

1 reference

Europe PubMed Central

PMC publication ID

23 June 2018

http://europepmc.org/abstract/PMC/5848659

sickle-cell disease

1 reference

22 April 2019

pathophysiology

1 reference

based on heuristic

inferred from title

Martin H Steinberg

1

1 reference

Europe PubMed Central

PMC publication ID

23 June 2018

http://europepmc.org/abstract/PMC/5848659

language of work or name

0 references

publication date

25 December 2008

2 references

Europe PubMed Central

PMC publication ID

23 June 2018

http://europepmc.org/abstract/PMC/5848659

22 April 2019

The Scientific World Journal

2 references

Europe PubMed Central

PMC publication ID

23 June 2018

http://europepmc.org/abstract/PMC/5848659

22 April 2019

8

1 reference

Europe PubMed Central

PMC publication ID

23 June 2018

http://europepmc.org/abstract/PMC/5848659

1295-1324

1 reference

Europe PubMed Central

PMC publication ID

23 June 2018

http://europepmc.org/abstract/PMC/5848659

copyright license

Creative Commons Attribution 3.0 Unported

1 reference

Europe PubMed Central

22 April 2019

copyright status

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on focus list of Wikimedia project

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Identifiers

10.1100/TSW.2008.157

1 reference

Europe PubMed Central

PMC publication ID

23 June 2018

http://europepmc.org/abstract/PMC/5848659

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

23 June 2018

http://europepmc.org/abstract/PMC/5848659

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

23 June 2018

http://europepmc.org/abstract/PMC/5848659

ResearchGate publication ID

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