(Q56904929)

1

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A W Bingaman

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M M Durham

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T C Pearson

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C P Larsen

Cells mediating allograft rejection.

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language of work or name

English

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publication date

October 2001

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published in

Transplant Immunology

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volume

9

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issue

1

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page(s)

13-7

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cites work

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Cells mediating graft rejection in the mouse. I. Lyt-1 cells mediate skin graft rejection

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Cells mediating graft rejection in the mouse. II. The Ly phenotypes of cells producing tumor allograft rejection

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A Theory of Self-Nonself Discrimination: Paralysis and induction involve the recognition of one and two determinants on an antigen, respectively

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T-cell antigen CD28 mediates adhesion with B cells by interacting with activation antigen B7/BB-1.

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Role of the CD28 receptor in T-cell activation

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CTLA-4 is a second receptor for the B cell activation antigen B7

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A new member of the immunoglobulin superfamily--CTLA-4

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Loss of CTLA-4 leads to massive lymphoproliferation and fatal multiorgan tissue destruction, revealing a critical negative regulatory role of CTLA-4

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Cytotoxic T lymphocyte antigen 4 (CTLA4) blockade accelerates the acute rejection of cardiac allografts in CD28-deficient mice: CTLA4 can function independently of CD28.

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Role of interleukin 12 and costimulators in T cell anergy in vivo.

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Long-term acceptance of skin and cardiac allografts after blocking CD40 and CD28 pathways

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Prolonged acceptance of concordant and discordant xenografts with combined CD40 and CD28 pathway blockade

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Induction of peripheral T cell tolerance in vivo requires CTLA-4 engagement.

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Determination of lymphocyte division by flow cytometry

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Analysis of the B7 costimulatory pathway in allograft rejection

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4-1BB costimulatory signals preferentially induce CD8+ T cell proliferation and lead to the amplification in vivo of cytotoxic T cell responses

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B7/CD28 costimulation is essential for the homeostasis of the CD4+CD25+ immunoregulatory T cells that control autoimmune diabetes

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