(Q58116631)

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Ceramide generated by acidic sphingomyelinase contributes to tumor necrosis factor-α-mediated apoptosis in human colon HT-29 cells through glycosphingolipids formation

scientific article published on 01 August 2002

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12 November 2019

Ceramide generated by acidic sphingomyelinase contributes to tumor necrosis factor-alpha-mediated apoptosis in human colon HT-29 cells through glycosphingolipids formation. Possible role of ganglioside GD3 (English)

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12 November 2019

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molecular biology

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structural biology

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apoptotic process

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based on heuristic

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12 November 2019

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12 November 2019

José C Fernández-Checa

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12 November 2019

Carmen Garcia-Ruiz

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Carmen García-Ruiz

4

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1 August 2002

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12 November 2019

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12 November 2019

526

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12 November 2019

1-3

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12 November 2019

135-141

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12 November 2019

Tumor necrosis factor's cytotoxic activity is signaled by the p55 TNF receptor

1 reference

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21 January 2018

Tumor necrosis factor receptors--structure and function

1 reference

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21 January 2018

Crystal structure of the soluble human 55 kd TNF receptor-human TNF beta complex: implications for TNF receptor activation

1 reference

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21 January 2018

Regulation of ceramide production and apoptosis

1 reference

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21 January 2018

Ceramide in the eukaryotic stress response

1 reference

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21 January 2018

Enzymes of sphingolipid metabolism: from modular to integrative signaling

1 reference

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21 January 2018

Ceramide generation by two distinct pathways in tumor necrosis factor alpha-induced cell death

1 reference

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21 January 2018

Zn2+-stimulated sphingomyelinase is secreted by many cell types and is a product of the acid sphingomyelinase gene

1 reference

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21 January 2018

Oocyte apoptosis is suppressed by disruption of the acid sphingomyelinase gene or by sphingosine-1-phosphate therapy

1 reference

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21 January 2018

Cell autonomous apoptosis defects in acid sphingomyelinase knockout fibroblasts.

1 reference

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21 January 2018

Overexpression of acid ceramidase protects from tumor necrosis factor-induced cell death

1 reference

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21 January 2018

Role of acidic sphingomyelinase in Fas/CD95-mediated cell death.

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2802%2903140-X

21 January 2018

Natural ceramide reverses Fas resistance of acid sphingomyelinase(-/-) hepatocytes.

1 reference

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21 January 2018

Acidic sphingomyelinase (ASM) is necessary for fas-induced GD3 ganglioside accumulation and efficient apoptosis of lymphoid cells

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2802%2903140-X

21 January 2018

CD95 (Fas/APO-1) induces ceramide formation and apoptosis in the absence of a functional acid sphingomyelinase.

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2802%2903140-X

21 January 2018

Cell adhesion/recognition and signal transduction through glycosphingolipid microdomain

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2802%2903140-X

21 January 2018

Commitment to apoptosis by GD3 ganglioside depends on opening of the mitochondrial permeability transition pore.

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2802%2903140-X

21 January 2018

GD3 ganglioside directly targets mitochondria in a bcl-2-controlled fashion

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2802%2903140-X

21 January 2018

Requirement for GD3 ganglioside in CD95- and ceramide-induced apoptosis.

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2802%2903140-X

21 January 2018

GD3 glycosphingolipid contributes to Fas-mediated apoptosis via association with ezrin cytoskeletal protein

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2802%2903140-X

21 January 2018

Interferon-gamma-induced differentiation and apoptosis of HT29 cells: dissociation of (glucosyl)ceramide signaling

1 reference

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21 January 2018

Drosophila grim induces apoptosis in mammalian cells.

1 reference

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21 January 2018

Alteration of ganglioside composition by stable transfection with antisense vectors against GD3-synthase gene expression

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2802%2903140-X

21 January 2018

GM3 ganglioside inhibits endothelin-1-mediated signal transduction in C6 glioma cells.

1 reference

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21 January 2018

Death receptors in liver biology and pathobiology

1 reference

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7 January 2021

Sphingolipid biosynthesis de novo by rat hepatocytes in culture. Ceramide and sphingomyelin are associated with, but not required for, very low density lipoprotein secretion

1 reference

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7 January 2021

Daunorubicin-induced apoptosis: triggering of ceramide generation through sphingomyelin hydrolysis

1 reference

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7 January 2021

Serine protease inhibitors block neutral sphingomyelinase activation, ceramide generation, and apoptosis triggered by daunorubicin

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2802%2903140-X

7 January 2021

Functional dichotomy of neutral and acidic sphingomyelinases in tumor necrosis factor signaling

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2802%2903140-X

7 January 2021

Compartmentalized Production of Ceramide at the Cell Surface

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2802%2903140-X

7 January 2021

Gangliosides turnover and neural cells function: a new perspective.

1 reference

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7 January 2021

Apoptogenic ganglioside GD3 directly induces the mitochondrial permeability transition

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2802%2903140-X

7 January 2021

The tricyclic antidepressant desipramine causes proteolytic degradation of lysosomal sphingomyelinase in human fibroblasts

1 reference

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7 January 2021

Extensive Glycosphingolipid Depletion in the Liver and Lymphoid Organs of Mice Treated withN-Butyldeoxynojirimycin

1 reference

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7 January 2021

Beta-amyloid-induced synthesis of the ganglioside GD3 is a requisite for cell cycle reactivation and apoptosis in neurons

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2802%2903140-X

7 January 2021

Direct interaction of GD3 ganglioside with mitochondria generates reactive oxygen species followed by mitochondrial permeability transition, cytochrome c release, and caspase activation

1 reference

https://api.crossref.org/works/10.1016%2FS0014-5793%2802%2903140-X

7 January 2021

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10.1016/S0014-5793(02)03140-X

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Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12208520%20AND%20SRC:MED&resulttype=core&format=json

12 November 2019

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:12208520%20AND%20SRC:MED&resulttype=core&format=json

12 November 2019

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