(Q58800404)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:21792878%20AND%20SRC:MED&resulttype=core&format=json

8 February 2020

title

SH2-domain mutations in STAT3 in hyper-IgE syndrome patients result in impairment of IL-10 function (English)

1 reference

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8 February 2020

1 reference

2

1 reference

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8 February 2020

Flavia Bazzoni

9

1 reference

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8 February 2020

Raffaele Badolato

10

1 reference

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8 February 2020

author name string

Mauro Giacomelli

1

1 reference

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8 February 2020

Daniele Moratto

3

1 reference

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8 February 2020

Patrizia Bertolini

4

1 reference

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8 February 2020

Giampaolo Ricci

5

1 reference

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8 February 2020

Cristina Bertulli

6

1 reference

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8 February 2020

Alessandro Plebani

7

1 reference

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8 February 2020

Marco Cassatella

8

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8 February 2020

language of work or name

English

0 references

publication date

6 September 2011

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:21792878%20AND%20SRC:MED&resulttype=core&format=json

European Journal of Immunology

8 February 2020

published in

1 reference

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8 February 2020

volume

41

1 reference

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8 February 2020

issue

10

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8 February 2020

page(s)

3075-3084

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:21792878%20AND%20SRC:MED&resulttype=core&format=json

A young man with hyperimmunoglobulin-E syndrome and IgA and IgG deficiencies

8 February 2020

cites work

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Metabolism of immunoglobulin E in patients with markedly elevated serum immunoglobulin E levels

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Hyper-IgE syndrome with recurrent infections--an autosomal dominant multisystem disorder

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Autosomal recessive hyperimmunoglobulin E syndrome: a distinct disease entity

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

STAT3 mutations in the hyper-IgE syndrome

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Dominant-negative mutations in the DNA-binding domain of STAT3 cause hyper-IgE syndrome

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Novel and recurrent STAT3 mutations in hyper-IgE syndrome patients from different ethnic groups.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Signaling through the JAK/STAT pathway, recent advances and future challenges

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

STATs and gene regulation

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Insights into the role of STAT3 in human lymphocyte differentiation as revealed by the hyper-IgE syndrome

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Interleukin-10 and the interleukin-10 receptor

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Interleukin 10(IL-10) inhibits cytokine synthesis by human monocytes: an autoregulatory role of IL-10 produced by monocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Interleukin 10 (IL-10) and viral IL-10 strongly reduce antigen-specific human T cell proliferation by diminishing the antigen-presenting capacity of monocytes via downregulation of class II major histocompatibility complex expression

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Interleukin-10 (IL-10) inhibits the induction of nitric oxide synthase by interferon-gamma in murine macrophages

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Uncovering an IL-10-dependent NF-κB recruitment to the IL-1ra promoter that is impaired in STAT3 functionally defective patients

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

An autoinflammatory disease with deficiency of the interleukin-1-receptor antagonist

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

An autoinflammatory disease due to homozygous deletion of the IL1RN locus

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Dendritic cell (DC)-specific targeting reveals Stat3 as a negative regulator of DC function

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Cutting edge: proangiogenic properties of alternatively activated dendritic cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Dendritic cells and the control of immunity

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Dendritic cells: specialized and regulated antigen processing machines

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Inflammatory stimuli induce accumulation of MHC class II complexes on dendritic cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Dendritic cell trafficking: more than just chemokines.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Functional defects of dendritic cells in patients with CD40 deficiency

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Mutations in STAT3 and diagnostic guidelines for hyper-IgE syndrome

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Up-regulation of IL-10R1 expression is required to render human neutrophils fully responsive to IL-10.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

IL-10 modulates cytokine gene transcription by protein synthesis-independent and dependent mechanisms in lipopolysaccharide-treated neutrophils

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Circulating neutrophils of septic patients constitutively express IL-10R1 and are promptly responsive to IL-10.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Molecular explanation for the contradiction between systemic Th17 defect and localized bacterial infection in hyper-IgE syndrome

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

New mechanism of oral immunity to mucosal candidiasis in hyper-IgE syndrome

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Defective IL-10 signaling in hyper-IgE syndrome results in impaired generation of tolerogenic dendritic cells and induced regulatory T cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Activation of an immunoregulatory and antiviral gene expression program in poly(I:C)-transfected human neutrophils

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

Job's Syndrome. Recurrent, "cold", staphylococcal abscesses

21 January 2018

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

inferred from DOI database lookup

7 January 2021

based on heuristic

Interleukin-10 differentially regulates cytokine inhibitor and chemokine release from blood mononuclear cells and fibroblasts

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

inferred from DOI database lookup

7 January 2021

based on heuristic

Effect of interleukin-10 on dendritic cell maturation and function

1 reference

https://api.crossref.org/works/10.1002%2FEJI.201141721

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1002/EJI.201141721

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:21792878%20AND%20SRC:MED&resulttype=core&format=json

8 February 2020

1 reference