(Q58860453)

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Myriadenolide, a labdane diterpene isolated from Alomia myriadenia (asteraceae) induces depolarization of mitochondrial membranes and apoptosis associated with activation of caspases-8, -9, and -3 in Jurkat and THP-1 cells

scientific article published on 01 November 2003

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20 December 2019

Myriadenolide, a labdane diterpene isolated from Alomia myriadenia (asteraceae) induces depolarization of mitochondrial membranes and apoptosis associated with activation of caspases-8, -9, and -3 in Jurkat and THP-1 cells (English)

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20 December 2019

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Carlos Leomar Zani

5

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20 December 2019

Elaine Maria de Souza-Fagundes

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Elaine Maria Souza-Fagundes

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20 December 2019

Gabriela Brumatti

2

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20 December 2019

Olindo Assis Martins-Filho

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20 December 2019

Rodrigo Corrêa-Oliveira

4

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20 December 2019

Gustavo Pessini Amarante-Mendes

6

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20 December 2019

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1 November 2003

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20 December 2019

Experimental Cell Research

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20 December 2019

290

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20 December 2019

2

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20 December 2019

420-426

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Biological activity of the labdane diterpenes

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7 January 2021

A cytotoxic diterpene from Alomia myriadenia

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A rapid and simple method for measuring thymocyte apoptosis by propidium iodide staining and flow cytometry

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Early redistribution of plasma membrane phosphatidylserine is a general feature of apoptosis regardless of the initiating stimulus: inhibition by overexpression of Bcl-2 and Abl

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Anti-apoptotic oncogenes prevent caspase-dependent and independent commitment for cell death

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Collapse of the inner mitochondrial transmembrane potential is not required for apoptosis of HL60 cells.

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The cytotoxic cell protease granzyme B initiates apoptosis in a cell-free system by proteolytic processing and activation of the ICE/CED-3 family protease, CPP32, via a novel two-step mechanism.

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Up-regulation of Fas (CD95) in human p53wild-type cancer cells treated with ionizing radiation

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Drug-induced apoptosis in hepatoma cells is mediated by the CD95 (APO-1/Fas) receptor/ligand system and involves activation of wild-type p53.

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Activation of the CD95 (APO-1/Fas) pathway in drug- and gamma-irradiation-induced apoptosis of brain tumor cells

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Involvement of the CD95 (APO–1/Fas) receptor/ligand system in drug–induced apoptosis in leukemia cells

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Deficient activation of the CD95 (APO-1/Fas) system in drug-resistant cells

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7 January 2021

Chemotherapeutic drug-induced apoptosis in human leukaemic cells is independent of the Fas (APO-1/CD95) receptor/ligand system.

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7 January 2021

FADD: essential for embryo development and signaling from some, but not all, inducers of apoptosis

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Fas ligand-independent, FADD-mediated activation of the Fas death pathway by anticancer drugs.

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Activation of caspase-8 in drug-induced apoptosis of B-lymphoid cells is independent of CD95/Fas receptor-ligand interaction and occurs downstream of caspase-3.

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Identifiers

10.1016/S0014-4827(03)00350-1

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:14567999%20AND%20SRC:MED&resulttype=core&format=json

20 December 2019

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:14567999%20AND%20SRC:MED&resulttype=core&format=json

20 December 2019

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