(Q59114615)

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Neuronal nitric oxide synthase is up-regulated by angiotensin II and attenuates NADPH oxidase activity and facilitates relaxation in murine left ventricular myocytes

scientific article published on 30 March 2012

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28 January 2020

Neuronal nitric oxide synthase is up-regulated by angiotensin II and attenuates NADPH oxidase activity and facilitates relaxation in murine left ventricular myocytes (English)

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28 January 2020

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28 January 2020

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Chun Zi Jin

1

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28 January 2020

Ji Hyun Jang

2

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28 January 2020

Yue Wang

3

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28 January 2020

Jae Gon Kim

4

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28 January 2020

Jun Shi

6

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28 January 2020

Cheng Ri Che

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28 January 2020

Sung Joon Kim

8

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28 January 2020

Yin Hua Zhang

9

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28 January 2020

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30 March 2012

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28 January 2020

Journal of Molecular and Cellular Cardiology

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28 January 2020

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28 January 2020

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28 January 2020

1274-1281

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28 January 2020

Local cardiac renin-angiotensin system: hypertension and cardiac failure

1 reference

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7 January 2021

Nox proteins in signal transduction

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7 January 2021

Nitric oxide/reactive oxygen species generation and nitroso/redox imbalance in heart failure: from molecular mechanisms to therapeutic implications.

1 reference

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7 January 2021

NADPH oxidases and cardiac remodelling

1 reference

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7 January 2021

Acute effects of nitric oxide on left ventricular relaxation and diastolic distensibility in humans. Assessment by bicoronary sodium nitroprusside infusion

1 reference

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7 January 2021

Paracrine and autocrine effects of nitric oxide on myocardial function

1 reference

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7 January 2021

Cardiac neuronal nitric oxide synthase isoform regulates myocardial contraction and calcium handling

1 reference

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7 January 2021

Reduced phospholamban phosphorylation is associated with impaired relaxation in left ventricular myocytes from neuronal NO synthase-deficient mice.

1 reference

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7 January 2021

Increased neuronal nitric oxide synthase-derived NO production in the failing human heart

1 reference

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7 January 2021

Role of myocardial neuronal nitric oxide synthase-derived nitric oxide in beta-adrenergic hyporesponsiveness after myocardial infarction-induced heart failure in rat.

1 reference

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7 January 2021

Up-regulation of cardiac nitric oxide synthase 1-derived nitric oxide after myocardial infarction in senescent rats.

1 reference

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7 January 2021

nNOS gene deletion exacerbates pathological left ventricular remodeling and functional deterioration after myocardial infarction

1 reference

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7 January 2021

Conditional overexpression of neuronal nitric oxide synthase is cardioprotective in ischemia/reperfusion.

1 reference

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7 January 2021

The role of nitric oxide and reactive oxygen species in the positive inotropic response to mechanical stretch in the mammalian myocardium

1 reference

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7 January 2021

A defect of neuronal nitric oxide synthase increases xanthine oxidase-derived superoxide anion and attenuates the control of myocardial oxygen consumption by nitric oxide derived from endothelial nitric oxide synthase

1 reference

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7 January 2021

Changes in NOS activity and protein expression during acute and prolonged ANG II administration

1 reference

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7 January 2021

Expression and distribution of NOS1 and NOS3 in the myocardium of angiotensin II-infused rats

1 reference

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7 January 2021

Angiotensin II-induced increase in myocardial distensibility and its modulation by the endocardial endothelium in the rabbit heart.

1 reference

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7 January 2021

Physiologic basis and pathophysiologic implications of the diastolic properties of the cardiac muscle

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7 January 2021

AT2 receptors: functional relevance in cardiovascular disease.

1 reference

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7 January 2021

NAD(P)H oxidase-derived hydrogen peroxide mediates endothelial nitric oxide production in response to angiotensin II.

1 reference

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7 January 2021

A nitric oxide/cysteine interaction mediates the activation of soluble guanylate cyclase

1 reference

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7 January 2021

Long-term effect of neuronal nitric oxide synthase over-expression on cardiac neurotransmission mediated by a lentiviral vector.

1 reference

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7 January 2021

Conditional neuronal nitric oxide synthase overexpression impairs myocardial contractility

1 reference

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7 January 2021

Specific role of neuronal nitric-oxide synthase when tethered to the plasma membrane calcium pump in regulating the beta-adrenergic signal in the myocardium

1 reference

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7 January 2021

Sildenafil and cardiomyocyte-specific cGMP signaling prevent cardiomyopathic changes associated with dystrophin deficiency

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7 January 2021

Pressure-overload-induced subcellular relocalization/oxidation of soluble guanylyl cyclase in the heart modulates enzyme stimulation

1 reference

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7 January 2021

Neuronal nitric oxide synthase signaling within cardiac myocytes targets phospholamban.

1 reference

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7 January 2021

Regulation by phosphodiesterase isoforms of protein kinase A-mediated attenuation of myocardial protein kinase D activation

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7 January 2021

Calcineurin inhibition normalizes beta-adrenergic responsiveness in the spontaneously hypertensive rat.

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7 January 2021

miR-1 overexpression enhances Ca(2+) release and promotes cardiac arrhythmogenesis by targeting PP2A regulatory subunit B56alpha and causing CaMKII-dependent hyperphosphorylation of RyR2

1 reference

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7 January 2021

Positive chronotropic and inotropic effects of angiotensin II in the dog heart

1 reference

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7 January 2021

Inotropic effects of angiotensin II on human cardiac muscle in vitro

1 reference

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7 January 2021

Species-related differences in inotropic effects of angiotensin II in mammalian ventricular muscle: receptors, subtypes and phosphoinositide hydrolysis

1 reference

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7 January 2021

Effects of angiotensin II on myocardial contractility during short-term pressor responses to angiotensin II

1 reference

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7 January 2021

Angiotensin II-induced negative inotropy in rat ventricular myocytes: role of reactive oxygen species and p38 MAPK.

1 reference

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7 January 2021

Developmental conversion of inotropism by endothelin I and angiotensin II from positive to negative in mice.

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Angiotensin II-mediated adaptive and maladaptive remodeling of cardiomyocyte excitation-contraction coupling

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Cardiac fibrosis in mice with hypertrophic cardiomyopathy is mediated by non-myocyte proliferation and requires Tgf-β.

1 reference

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7 January 2021

TGF-beta1 improves cardiac performance via up-regulation of laminin receptor 37/67 in adult ventricular cardiomyocytes

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7 January 2021

Identifiers

10.1016/J.YJMCC.2012.03.013

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:22484619%20AND%20SRC:MED&resulttype=core&format=json

28 January 2020

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:22484619%20AND%20SRC:MED&resulttype=core&format=json

28 January 2020

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