(Q71977974)

English

Early spontaneous intermittent myocardial reperfusion during acute myocardial infarction is associated with augmented thrombogenic activity and less myocardial damage

scientific article published on 01 September 1995

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22 October 2019

Early spontaneous intermittent myocardial reperfusion during acute myocardial infarction is associated with augmented thrombogenic activity and less myocardial damage (English)

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22 October 2019

acute myocardial infarction

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based on heuristic

inferred from title

myocardial damage

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based on heuristic

inferred from title

author name string

A. W. Haider

1

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8 July 2024

F. Andreotti

2

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8 July 2024

D. R. Hackett

3

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8 July 2024

D. Tousoulis

4

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8 July 2024

C. Kluft

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8 July 2024

A. Maseri

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8 July 2024

G. J. Davies

7

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8 July 2024

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1 September 1995

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22 October 2019

full work available at URL

https://doi.org/10.1016/0735-1097(95)00210-u

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8 July 2024

https://api.elsevier.com/content/article/PII:073510979500210U?httpAccept=text/xml

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10.1016/0735-1097(95)00210-U

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8 July 2024

https://api.elsevier.com/content/article/PII:073510979500210U?httpAccept=text/plain

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10.1016/0735-1097(95)00210-U

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8 July 2024

Journal of the American College of Cardiology

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22 October 2019

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22 October 2019

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22 October 2019

662-667

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22 October 2019

Prevalence of total coronary occlusion during the early hours of transmural myocardial infarction

1

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Prevention of myocardial infarction by very early treatment with intracoronary streptokinase. Some clinical observations

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Intermittent coronary occlusion in acute myocardial infarction. Value of combined thrombolytic and vasodilator therapy

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Importance of reperfusion on thromboxane A2 metabolite excretion after thrombolysis

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Activation of prothrombin accompanying thrombolysis with recombinant tissue-type plasminogen activator

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Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium

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Myocardial protection with preconditioning

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Protection against infarction afforded by preconditioning is mediated by A1 adenosine receptors in rabbit heart

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Adaptation to ischemia during percutaneous transluminal coronary angioplasty. Clinical, hemodynamic, and metabolic features

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Preconditioning the human myocardium

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An automated fibrinolytic assay performed in microtiter plates

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Electrocardiographic prediction of coronary artery patency after thrombolytic treatment in acute myocardial infarction: use of the ST segment as a non-invasive marker

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Preservation of global and regional left ventricular function after early thrombolysis in acute myocardial infarction

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Reasons for the lack of benefit of immediate angioplasty during recombinant tissue plasminogen activator therapy for acute myocardial infarction: a regional wall motion analysis. European Cooperative Study Group

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Quantitative assessment of the extent of myocardial infarction in the conscious dog by means of analysis of serial changes in serum creatine phosphokinase activity

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Estimation of infarct size from serum MB creatine phosphokinase activity: Applications and limitations

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Relation between electrocardiographic and enzymatic methods of estimating acute myocardial infarct size

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Early coronary reperfusion blunts the procoagulant response of plasminogen activator inhibitor-1 and von Willebrand factor in acute myocardial infarction

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Residual flow to the infarct zone as a determinant of infarct size after direct angioplasty

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Improved myocardial ischemic response and enhanced collateral circulation with long repetitive coronary occlusion during angioplasty: a prospective study

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Timing of coronary recanalization. Paradigms, paradoxes, and pertinence

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The unstable ST segment early after thrombolysis for acute infarction and its usefulness as a marker of recurrent coronary occlusion

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Dynamic QRS complex and ST segment vectorcardiographic monitoring can identify vessel patency in patients with acute myocardial infarction treated with reperfusion therapy

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Generation of the combined prothrombin activation peptide (F1-2) during the clotting of blood and plasma

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Clinical significance of "circulating fibrin monomers'.

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Clot-bound thrombin is protected from inhibition by heparin-antithrombin III but is susceptible to inactivation by antithrombin III-independent inhibitors

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The pathogenesis of coronary artery disease and the acute coronary syndromes (2).

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Augmentation of plasminogen activator inhibitor type 1 activity in plasma by thrombosis and by thrombolysis

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Adrenergic mechanisms in the systemic plasminogen active response to adrenaline in man

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Importance of continued activation of thrombin reflected by fibrinopeptide A to the efficacy of thrombolysis

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Induction of marked thrombin activity by pharmacologic concentrations of plasminogen activators in nonanticoagulated whole blood

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10.1016/0735-1097(95)00210-U

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Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:7642856%20AND%20SRC:MED&resulttype=core&format=json

22 October 2019

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:7642856%20AND%20SRC:MED&resulttype=core&format=json

22 October 2019

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