(Q72868719)

English

Slow interaction of islet-activating protein with pancreatic islets during primary culture to cause reversal of alpha-adrenergic inhibition of insulin secretion

scientific article published on October 25, 1980

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scholarly article

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29 October 2019

Slow interaction of islet-activating protein with pancreatic islets during primary culture to cause reversal of alpha-adrenergic inhibition of insulin secretion (English)

1 reference

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29 October 2019

1 reference

10.1016/S0021-9258(18)43431-X

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8 September 2023

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author name string

T. Katada

1

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8 September 2023

M. Ui

2

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publication date

1 October 1980

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29 October 2019

25 October 1980

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National Center for Biotechnology Information

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8 September 2023

full work available at URL

http://intl.jbc.org/cgi/content/abstract/255/20/9580

online access status

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8 September 2023

https://api.elsevier.com/content/article/PII:S002192581843431X?httpAccept=text/xml

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https://api.elsevier.com/content/article/PII:S002192581843431X?httpAccept=text/plain

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Journal of Biological Chemistry

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29 October 2019

255

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29 October 2019

9580-9588

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29 October 2019

20

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29 October 2019

Biological Properties of Islets-Activating Protein (IAP) Purified from the Culture Medium of Bordetella pertussis

2

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10.1016/S0021-9258(18)43431-X

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8 September 2023

Islet activating protein (IAP) derived from the culture supernatant fluid of Bordetella pertussis: Effect on spontaneous diabetic rats

3

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Effect of in Vivo Pretreatment of Rats with a New Protein Purified fromBordetellapertussis onin VitroSecretion of Insulin: Role of Calcium

4

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Potentiation of the Adrenergic Beta-Receptor-Mediated Insulin Secretion in Pertussis-Sensitized Rats

6

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Accelerated turnover of blood glucose in pertussis-sensitized rats due to combined actions of endogenous insulin and adrenergic beta-stimulation

7

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Spontaneous recovery from streptozotocin-induced diabetes in rats pretreated with pertussis vaccine or hydrocortisone

8

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Perfusion of the Pancreas Isolated from Pertussis-Sensitized Rats: Potentiation of Insulin Secretory Responses Due to β-Adrenergic Stimulation

9

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Attenuation of epinephrine-induced increase in liver cyclic AMP by endogenous insulin in vivo

10

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Method for the Isolation of Intact Islets of Langerhans from the Rat Pancreas

11

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Coated Charcoal Immunoassay of Insulin

12

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An ultrasensitive method for the simultaneous determination of cyclic AMP and cyclic GMP in small-volume samples from blood and tissue

13

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The Stimulus-Secretion Coupling of Glucose-Induced Insulin Release. XIV. Glucose Regulation of Insular Biosynthetic Activity1

14

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Preservation of beta cell function in adult human pancreatic islets for several months in vitro.

15

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Coupling of insulin receptors to glucose transport: A temperature-dependent time lag in activation of glucose transport

16

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The Stimulus-secretion Coupling of Glucose-induced Insulin Release: V. The Participation of a Microtubular-microfilamentous System

17

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Perifusion of Isolated Rat Islets in Vitro: Participation of the Microtubular System in the Biphasic Release of Insulin

18

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Dynamics of Insulin Release and Microtubular-Microfilamentous System. I. EFFECT OF CYTOCHALASIN B

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Dynamics of Insulin Release and Microtubular-microfilamentous System: VII. Do Microfilaments Provide the Motive Force for the Translocation and Extrusion of Beta Granules?

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The glucagon receptor of rat liver plasma membrane can couple to adenylate cyclase without activating it.

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The lipid environment of the glucagon receptor regulates adenylate cyclase activity

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New Hypothesis of Insulin Secretion

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Involvement of nicotinamide adenine dinucleotide in the action of cholera toxin in vitro

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Effect of cholera enterotoxin on ion transport across isolated ileal mucosa

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Cholera toxin-fat cell interaction and the mechanism of activation of the lipolytic response.

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Mechanism of activation of adenylate cyclase by Vibrio cholerae enterotoxin

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Identifiers

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Europe PubMed Central

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29 October 2019

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