(Q73270557)

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Amelioration of relapsing experimental autoimmune encephalomyelitis with altered myelin basic protein peptides involves different cellular mechanisms

scientific article published on 01 April 1997

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scholarly article

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2 November 2019

Amelioration of relapsing experimental autoimmune encephalomyelitis with altered myelin basic protein peptides involves different cellular mechanisms (English)

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2 November 2019

experimental autoimmune encephalomyelitis

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author name string

A Gaur

1

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2 November 2019

S A Boehme

2

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2 November 2019

D Chalmers

3

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2 November 2019

P D Crowe

4

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2 November 2019

A Pahuja

5

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2 November 2019

N Ling

6

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2 November 2019

S Brocke

7

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2 November 2019

L Steinman

8

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2 November 2019

P J Conlon

9

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2 November 2019

publication date

1 April 1997

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2 November 2019

Journal of Neuroimmunology

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2 November 2019

74

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2 November 2019

149-158

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2 November 2019

1-2

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2 November 2019

Die and let live: eliminating dangerous lymphocytes

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Treatment of experimental encephalomyelitis with a peptide analogue of myelin basic protein

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T cell deletion in high antigen dose therapy of autoimmune encephalomyelitis

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Peptide-specific prevention of experimental allergic encephalomyelitis. Neonatal tolerance induced to the dominant T cell determinant of myelin basic protein

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Separation of IL-4 production from Th cell proliferation by an altered T cell receptor ligand

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Induction of TH1 and TH2 immunity in neonatal mice.

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T cell receptor antagonist peptides are highly effective inhibitors of experimental allergic encephalomyelitis

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Amelioration of Autoimmune Encephalomyelitis by Myelin Basic Protein Synthetic Peptide-Induced Anergy

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Immunotherapeutic strategies directed at the trimolecular complex.

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T cell receptor antagonist peptides induce positive selection

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Development of TH1 CD4+ T cells through IL-12 produced by Listeria-induced macrophages

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Reversal of experimental autoimmune encephalomyelitis by a soluble peptide variant of a myelin basic protein epitope: T cell receptor antagonism and reduction of interferon gamma and tumor necrosis factor alpha production

1 reference

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An altered peptide ligand mediates immune deviation and prevents autoimmune encephalomyelitis

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Selection for T-cell receptor V beta-D beta-J beta gene rearrangements with specificity for a myelin basic protein peptide in brain lesions of multiple sclerosis

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T-cell recognition of an immuno-dominant myelin basic protein epitope in multiple sclerosis

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Negative selection of CD4+ CD8+ thymocytes by T-cell receptor peptide antagonists

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Lymphocyte responses and cytokines

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Altered peptide ligands can control CD4 T lymphocyte differentiation in vivo

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Cytokine-induced immune deviation as a therapy for inflammatory autoimmune disease

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The T cell receptor repertoire influences V beta element usage in response to myoglobin.

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Involvement of distinct murine T-cell receptors in the autoimmune encephalitogenic response to nested epitopes of myelin basic protein

1 reference

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Characterization of a major encephalitogenic T cell epitope in SJL/J mice with synthetic oligopeptides of myelin basic protein

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Prevention of experimental encephalomyelitis with peptides that block interaction of T cells with major histocompatibility complex proteins

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Induction of T-cell anergy by altered T-cell-receptor ligand on live antigen-presenting cells

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Th2 cell clonal anergy as a consequence of partial activation

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A single amino acid change in a myelin basic protein peptide confers the capacity to prevent rather than induce experimental autoimmune encephalomyelitis

1 reference

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Major T-cell responses in multiple sclerosis.

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IL4 dictates T-cell differentiation

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Binding of myelin basic protein peptides to human histocompatibility leukocyte antigen class II molecules and their recognition by T cells from multiple sclerosis patients

1 reference

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Modulation of cytokine patterns of human autoreactive T cell clones by a single amino acid substitution of their peptide ligand

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Antigen recognition in autoimmune encephalomyelitis and the potential for peptide-mediated immunotherapy.

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The T lymphocyte in experimental allergic encephalomyelitis

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Increased frequency of interleukin 2-responsive T cells specific for myelin basic protein and proteolipid protein in peripheral blood and cerebrospinal fluid of patients with multiple sclerosis

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Identifiers

10.1016/S0165-5728(96)00220-2

1 reference

Europe PubMed Central

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2 November 2019

1 reference

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2 November 2019

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