(Q73605805)

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Intrathecal interleukin-1 receptor antagonist in combination with soluble tumor necrosis factor receptor exhibits an anti-allodynic action in a rat model of neuropathic pain

scientific article published on 01 January 2001

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5 November 2019

Intrathecal interleukin-1 receptor antagonist in combination with soluble tumor necrosis factor receptor exhibits an anti-allodynic action in a rat model of neuropathic pain (English)

1 reference

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5 November 2019

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S Sweitzer

1

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5 November 2019

D Martin

2

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5 November 2019

J A DeLeo

3

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1 January 2001

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5 November 2019

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5 November 2019

103

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5 November 2019

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5 November 2019

529-539

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5 November 2019

Increase of interleukin-6 mRNA in the spinal cord following peripheral nerve injury in the rat: potential role of IL-6 in neuropathic pain.

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Effects of interleukin-1 receptor antagonist in a slow-release hylan vehicle on rat type II collagen arthritis

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Synergy between tumor necrosis factor alpha and interleukin-1 in the induction of sickness behavior in mice.

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Dissociation of microglial activation and neuropathic pain behaviors following peripheral nerve injury in the rat.

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Microtiter immunocytochemical ELISA assay

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Interleukin-6-mediated hyperalgesia/allodynia and increased spinal IL-6 expression in a rat mononeuropathy model

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Cytokine and growth factor immunohistochemical spinal profiles in two animal models of mononeuropathy.

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Transgenic expression of TNF by astrocytes increases mechanical allodynia in a mouse neuropathy model

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Gender differences in rat neuropathic pain sensitivity is dependent on strain

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Tumor necrosis factor-alpha induces substance P in sympathetic ganglia through sequential induction of interleukin-1 and leukemia inhibitory factor

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PEGylated recombinant human soluble tumour necrosis factor receptor type I (r-Hu-sTNF-RI): novel high affinity TNF receptor designed for chronic inflammatory diseases

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Tumor necrosis factor alpha inhibits glutamate uptake by primary human astrocytes. Implications for pathogenesis of HIV-1 dementia

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Gender difference in analgesic response to the kappa-opioid pentazocine

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Kappa-opioids produce significantly greater analgesia in women than in men.

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Cytokines and the nervous system. I: Expression and recognition

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Interleukin-1 beta induces long-term increase of axonally transported opiate receptors and substance P

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Kidney as a major clearance organ for recombinant human interleukin-1 receptor antagonist

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Involvement of corticotropin-releasing factor in the antinociception produced by interleukin-1 in mice.

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Tumour necrosis factor-alpha increases intracellular Ca2+ and induces a depolarization in cultured astroglial cells.

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Prolonged ovarian sex steroid treatment of male rats produces antinociception: identification of sex-based divergent analgesic mechanisms

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7 January 2021

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The possible role of glia in nociceptive processing and hyperalgesia in the spinal cord of the rat

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7 January 2021

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Endogenous interleukin-6 contributes to hypersensitivity to cutaneous stimuli and changes in neuropeptides associated with chronic nerve constriction in mice

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7 January 2021

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Intracerebroventricular injection of interleukin-1 beta induces hyperalgesia in rats

1 reference

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7 January 2021

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Intracerebroventricular injection of tumor necrosis factor-alpha induces thermal hyperalgesia in rats.

1 reference

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7 January 2021

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Intracerebroventricular injection of interleukin-6 induces thermal hyperalgesia in rats

1 reference

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Neuroglial activation repertoire in the injured brain: graded response, molecular mechanisms and cues to physiological function

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7 January 2021

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Spinal nerve lesion-induced mechanoallodynia and adrenergic sprouting in sensory ganglia are attenuated in interleukin-6 knockout mice

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Cytokines and the nervous system II: Actions and mechanisms of action

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Contribution of interleukin‐1β to the inflammation‐induced increase in nerve growth factor levels and inflammatory hyperalgesia

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Cascading glia reactions: a common pathomechanism and its differentiated control by cyclic nucleotide signaling

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Cytosolic domain of the type I interleukin-1 receptor spontaneously recruits signaling molecules to activate a proinflammatory gene

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Neutralizing antibodies to interleukin 1-receptor reduce pain associated behavior in mice with experimental neuropathy

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Hyperalgesia in experimental neuropathy is dependent on the TNF receptor 1

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Spinal interleukin-1beta reduces inflammatory pain.

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Acute peripheral inflammation induces moderate glial activation and spinal IL-1beta expression that correlates with pain behavior in the rat.

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Possible involvement of interleukin-1 in cyclooxygenase-2 induction after spinal cord injury in rats

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Gender variations in clinical pain experience

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Endoneurial injection of TNF-alpha produces neuropathic pain behaviors.

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Anti-inflammatory interleukin-10 therapy in CCI neuropathy decreases thermal hyperalgesia, macrophage recruitment, and endoneurial TNF-alpha expression.

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Mechanisms of tumor necrosis factor-alpha (TNF-alpha) hyperalgesia

1 reference

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Evidence for the involvement of spinal cord glia in subcutaneous formalin induced hyperalgesia in the rat.

1 reference

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1 reference

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1 reference

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7 January 2021

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Biphasic effects of intracerebroventricular interleukin-1 beta on mechanical nociception in the rat.

1 reference

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7 January 2021

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Identifiers

10.1016/S0306-4522(00)00574-1

1 reference

Europe PubMed Central

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5 November 2019

1 reference

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5 November 2019

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