(Q74178691)

English

Cerebrospinal fluid hypoxanthine, xanthine and uric acid levels may reflect glutamate-mediated excitotoxicity in different neurological diseases

scientific article published on 01 November 1997

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scholarly article

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9 November 2019

Cerebrospinal fluid hypoxanthine, xanthine and uric acid levels may reflect glutamate-mediated excitotoxicity in different neurological diseases (English)

1 reference

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9 November 2019

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author name string

J F Stover

1

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9 November 2019

K Lowitzsch

2

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9 November 2019

O S Kempski

3

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9 November 2019

publication date

1 November 1997

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9 November 2019

Neuroscience Letters

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9 November 2019

238

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9 November 2019

1-2

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9 November 2019

25-28

1 reference

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9 November 2019

Oxygen free radicals regulate NMDA receptor function via a redox modulatory site

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2897%2900840-9

7 January 2021

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Identification of hypoxanthine transport and xanthine oxidase activity in brain capillaries.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2897%2900840-9

7 January 2021

based on heuristic

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Differential distribution of purine metabolizing enzymes between glia and neurons

1 reference

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7 January 2021

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Oxidative stress, glutamate, and neurodegenerative disorders

1 reference

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7 January 2021

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Relationships among ATP synthesis, K+ gradients, and neurotransmitter amino acid levels in isolated rat brain synaptosomes.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2897%2900840-9

7 January 2021

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Patterns of increased glucose use following extracellular infusion of glutamate: an autoradiographic study

1 reference

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7 January 2021

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Blockade of N-methyl-D-aspartate-sensitive acidic amino acid receptors inhibits ischemia-induced accumulation of purine catabolites in the rat striatum.

1 reference

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7 January 2021

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Evidence for differential function of neuronal and glial cells in protein metabolism and amino acid transport

1 reference

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7 January 2021

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Interstitial and cerebrospinal fluid levels of energy-related metabolites after middle cerebral artery occlusion in rats

1 reference

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7 January 2021

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Effect of allopurinol on ischemia and reperfusion-induced cerebral injury in spontaneously hypertensive rats.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2897%2900840-9

7 January 2021

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Allopurinol inhibits uric acid accumulation in the rat brain following focal cerebral ischemia

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2897%2900840-9

7 January 2021

based on heuristic

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Adenosine triphosphate depletion reverses sodium-dependent, neuronal uptake of glutamate in rat hippocampal slices.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2897%2900840-9

7 January 2021

based on heuristic

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Glutamate becomes neurotoxic via the N-methyl-D-aspartate receptor when intracellular energy levels are reduced

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2897%2900840-9

7 January 2021

based on heuristic

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Ischemia-induced accumulation of extracellular amino acids in cerebral cortex, white matter, and cerebrospinal fluid.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2897%2900840-9

7 January 2021

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Ultrastructural immunocytochemical observations on the localization, metabolism and transport of glutamate in normal and ischemic brain tissue.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2897%2900840-9

7 January 2021

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Neurotransmitters in cerebrospinal fluid reflect pathological activity.

1 reference

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7 January 2021

based on heuristic

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Glutamate-induced intracellular acidification of cultured hippocampal neurons demonstrates altered energy metabolism resulting from Ca2+ loads

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2897%2900840-9

7 January 2021

based on heuristic

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Current concepts of excitotoxicity.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2897%2900840-9

7 January 2021

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inferred from DOI database lookup

Interaction between free radicals and excitatory amino acids in the blood-brain barrier disruption after iron injury in the rat.

1 reference

https://api.crossref.org/works/10.1016%2FS0304-3940%2897%2900840-9

7 January 2021

based on heuristic

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Identifiers

10.1016/S0304-3940(97)00840-9

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9464646%20AND%20SRC:MED&resulttype=core&format=json

9 November 2019

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9464646%20AND%20SRC:MED&resulttype=core&format=json

9 November 2019

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