(Q77384408)

English

Oral and subcutaneous administration of the glycosaminoglycan C3 attenuates Abeta(25-35)-induced abnormal tau protein immunoreactivity in rat brain

scientific article published on 01 January 2002

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scholarly article

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5 December 2019

Oral and subcutaneous administration of the glycosaminoglycan C3 attenuates Abeta(25-35)-induced abnormal tau protein immunoreactivity in rat brain (English)

1 reference

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5 December 2019

author name string

B Dudas

1

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5 December 2019

U Cornelli

2

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5 December 2019

J M Lee

3

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5 December 2019

M J Hejna

4

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5 December 2019

M Walzer

5

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5 December 2019

S A Lorens

6

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5 December 2019

R F Mervis

7

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5 December 2019

J Fareed

8

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5 December 2019

I Hanin

9

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5 December 2019

publication date

1 January 2002

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5 December 2019

Neurobiology of Aging

1 reference

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5 December 2019

23

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5 December 2019

1

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5 December 2019

97-104

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5 December 2019

Alzheimer disease amyloid beta protein forms calcium channels in bilayer membranes: blockade by tromethamine and aluminum

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beta-Amyloid peptide free radical fragments initiate synaptosomal lipoperoxidation in a sequence-specific fashion: implications to Alzheimer's disease.

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Perlecan binds to the beta-amyloid proteins (A beta) of Alzheimer's disease, accelerates A beta fibril formation, and maintains A beta fibril stability.

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Scrapie associated PrP accumulation and its prevention: insights from cell culture

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Glycosaminoglycan polysulfate (Ateroid) in old-age dementias: effects upon depressive symptomatology in geriatric patients

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Chondroitin sulfate proteoglycans are associated with the lesions of Alzheimer's disease

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The acute neurotoxicity and effects upon cholinergic axons of intracerebrally injected beta-amyloid in the rat brain

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Radiochemical micro assays for the determination of choline acetyltransferase and acetylcholinesterase activities

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7 January 2021

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Effects of injected Alzheimer beta-amyloid cores in rat brain.

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7 January 2021

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Similarities between beta amyloid peptides 1-40 and 40-1: effects on aggregation, toxicity in vitro, and injection in young and aged rats.

1 reference

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7 January 2021

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Differential effects of amyloid peptides beta-(1-40) and beta-(25-35) injections into the rat nucleus basalis.

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Assembly of microtubule-associated protein tau into Alzheimer-like filaments induced by sulphated glycosaminoglycans.

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Proteoglycan-mediated inhibition of A beta proteolysis. A potential cause of senile plaque accumulation.

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Alzheimer-like changes in microtubule-associated protein Tau induced by sulfated glycosaminoglycans. Inhibition of microtubule binding, stimulation of phosphorylation, and filament assembly depend on the degree of sulfation.

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A model for beta-amyloid aggregation and neurotoxicity based on free radical generation by the peptide: relevance to Alzheimer disease

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Aggregation state and neurotoxic properties of Alzheimer beta-amyloid peptide

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Arresting amyloidosis in vivo using small-molecule anionic sulphonates or sulphates: implications for Alzheimer's disease

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7 January 2021

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Beta-amyloid protein increases the vulnerability of cultured cortical neurons to excitotoxic damage

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7 January 2021

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The carboxyl third of tau is tightly bound to paired helical filaments.

1 reference

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7 January 2021

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An in vivo model for the neurodegenerative effects of beta amyloid and protection by substance P

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7 January 2021

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In vivo neurotoxicity of beta-amyloid [beta(1-40)] and the beta(25-35) fragment

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7 January 2021

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Phosphate analysis and dephosphorylation of modified tau associated with paired helical filaments

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7 January 2021

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Beta-amyloid neurotoxicity requires fibril formation and is inhibited by congo red.

1 reference

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7 January 2021

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Antigenic changes similar to those seen in neurofibrillary tangles are elicited by glutamate and Ca2+ influx in cultured hippocampal neurons

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beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicity

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Calcium-destabilizing and neurodegenerative effects of aggregated beta-amyloid peptide are attenuated by basic FGF.

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Amyloid plaque core protein in Alzheimer disease and Down syndrome

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7 January 2021

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Neurotoxicity of human amylin in rat primary hippocampal cultures: similarity to Alzheimer's disease amyloid-beta neurotoxicity

1 reference

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7 January 2021

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Peptide compositions of the cerebrovascular and senile plaque core amyloid deposits of Alzheimer's disease.

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7 January 2021

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Phosphorylation determines two distinct species of Tau in the central nervous system.

1 reference

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7 January 2021

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Neurodegeneration induced by beta-amyloid peptides in vitro: the role of peptide assembly state

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Aggregation of secreted amyloid beta-protein into sodium dodecyl sulfate-stable oligomers in cell culture

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Sulfated glycosaminoglycans and dyes attenuate the neurotoxic effects of beta-amyloid in rat PC12 cells.

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Beta-amyloid from Alzheimer disease brains inhibits sprouting and survival of sympathetic neurons.

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beta-Amyloid-(1-42) is a major component of cerebrovascular amyloid deposits: implications for the pathology of Alzheimer disease

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1 reference

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1 reference

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7 January 2021

based on heuristic

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1 reference

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Tau 2: a probe for a Ser conformation in the amino terminus of tau

1 reference

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1 reference

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Identifiers

10.1016/S0197-4580(01)00255-X

1 reference

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5 December 2019

1 reference

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5 December 2019

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