(Q77830046)

English

Lipopolysaccharide tolerance in murine peritoneal macrophages induces downregulation of the lipopolysaccharide signal transduction pathway through mitogen-activated protein kinase and nuclear factor-kappaB cascades, but not lipopolysaccharide-incorp

scientific article published on 01 June 1999

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scholarly article

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8 December 2019

Lipopolysaccharide tolerance in murine peritoneal macrophages induces downregulation of the lipopolysaccharide signal transduction pathway through mitogen-activated protein kinase and nuclear factor-kappaB cascades, but not lipopolysaccharide-incorporation steps (English)

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8 December 2019

lipopolysaccharide

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K Tominaga

1

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8 December 2019

S Saito

2

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8 December 2019

M Matsuura

3

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8 December 2019

M Nakano

4

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8 December 2019

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1 June 1999

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8 December 2019

Biochimica et Biophysica Acta

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8 December 2019

1450

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8 December 2019

130-144

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8 December 2019

2

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8 December 2019

Gram-negative endotoxin: an extraordinary lipid with profound effects on eukaryotic signal transduction.

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Receptor-dependent mechanisms of cell stimulation by bacterial endotoxin

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Endotoxin signal transduction in macrophages.

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Bacterial lipopolysaccharide stimulates protein tyrosine phosphorylation in macrophages

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Tyrosine phosphorylation of mitogen-activated protein kinases is necessary for activation of murine macrophages by natural and synthetic bacterial products

1 reference

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Lipopolysaccharide-induced cytokine production in human monocytes: role of tyrosine phosphorylation in transmembrane signal transduction

1 reference

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Prevention of lipopolysaccharide-induced lethal toxicity by tyrosine kinase inhibitors

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Radicicol, a protein tyrosine kinase inhibitor, suppresses the expression of mitogen-inducible cyclooxygenase in macrophages stimulated with lipopolysaccharide and in experimental glomerulonephritis

1 reference

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Protein-tyrosine kinase activation is required for lipopolysaccharide induction of interleukin 1beta and NFkappaB activation, but not NFkappaB nuclear translocation

1 reference

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A MAP kinase targeted by endotoxin and hyperosmolarity in mammalian cells

1 reference

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A protein kinase involved in the regulation of inflammatory cytokine biosynthesis

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Activation of c-Jun N-terminal kinase in bacterial lipopolysaccharide-stimulated macrophages

1 reference

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Nuclear factor kappa B, a mediator of lipopolysaccharide effects

1 reference

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Rel/NF-kappa B/I kappa B family: intimate tales of association and dissociation

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Multistep regulation mechanisms for tolerance induction to lipopolysaccharide lethality in the tumor-necrosis-factor-alpha-mediated pathway. Application of non-toxic monosaccharide lipid A analogues for elucidation of mechanisms

1 reference

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LPS pretreatment reprograms macrophage LPS-stimulated TNF and IL-1 release without protein tyrosine kinase activation

1 reference

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CD14, a receptor for complexes of lipopolysaccharide (LPS) and LPS binding protein

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Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

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Activation of interleukin-6 gene expression through the NF-kappa B transcription factor

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7 January 2021

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The preparation and characterization of a radioiodinated bacterial lipopolysaccharide

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7 January 2021

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Shared actions of endotoxin and taxol on TNF receptors and TNF release

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7 January 2021

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Independent human MAP-kinase signal transduction pathways defined by MEK and MKK isoforms

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7 January 2021

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Phosphorylation at threonine-235 by a ras-dependent mitogen-activated protein kinase cascade is essential for transcription factor NF-IL6

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7 January 2021

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Activation of the interleukin 6 gene by Mycobacterium tuberculosis or lipopolysaccharide is mediated by nuclear factors NF-IL6 and NF-kappa B.

1 reference

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7 January 2021

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Activation of the transcription factor MEF2C by the MAP kinase p38 in inflammation

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7 January 2021

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Lipopolysaccharide antagonists block taxol-induced signaling in murine macrophages

1 reference

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7 January 2021

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Paclitaxel (Taxol)-induced NF-kappaB translocation in murine macrophages.

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Identifiers

10.1016/S0167-4889(99)00037-3

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Europe PubMed Central

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8 December 2019

1 reference

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8 December 2019

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