(Q79325476)

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Troponin phosphorylation and regulatory function in human heart muscle: dephosphorylation of Ser23/24 on troponin I could account for the contractile defect in end-stage heart failure

scientific article published on 01 November 2006

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scholarly article

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21 December 2019

Troponin phosphorylation and regulatory function in human heart muscle: dephosphorylation of Ser23/24 on troponin I could account for the contractile defect in end-stage heart failure (English)

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21 December 2019

author name string

Andrew E Messer

1

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21 December 2019

Adam M Jacques

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21 December 2019

Steven B Marston

3

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1 November 2006

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21 December 2019

Journal of Molecular and Cellular Cardiology

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247-259

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21 December 2019

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21 December 2019

The failing human heart

1 reference

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Myofibrillar adenosine triphosphatase activity in congestive heart failure

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Alterations of cross-bridge kinetics in human atrial and ventricular myocardium

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Changes in myofibrillar content and Mg-ATPase activity in ventricular tissues from patients with heart failure caused by coronary artery disease, cardiomyopathy, or mitral valve insufficiency

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Myofibrillar calcium sensitivity of isometric tension is increased in human dilated cardiomyopathies: role of altered beta-adrenergically mediated protein phosphorylation

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Myofilament calcium regulation in human myocardium

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7 January 2021

Increased Ca2+-sensitivity of the contractile apparatus in end-stage human heart failure results from altered phosphorylation of contractile proteins

1 reference

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Effects of calcium, inorganic phosphate, and pH on isometric force in single skinned cardiomyocytes from donor and failing human hearts

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In vitro motility analysis of thin filaments from failing and non-failing human heart: troponin from failing human hearts induces slower filament sliding and higher Ca(2+) sensitivity

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Modulation of thin filament activation by breakdown or isoform switching of thin filament proteins: physiological and pathological implications

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Troponin I phosphorylation in the normal and failing adult human heart

1 reference

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Protein kinase A (PKA)-dependent troponin-I phosphorylation and PKA regulatory subunits are decreased in human dilated cardiomyopathy

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Increased protein kinase C activity and expression of Ca2+-sensitive isoforms in the failing human heart

1 reference

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7 January 2021

Regulation of cardiac contractile function by troponin I phosphorylation

1 reference

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7 January 2021

Decreased protein and phosphorylation level of the protein phosphatase inhibitor-1 in failing human hearts

1 reference

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7 January 2021

Microanalysis and distribution of cardiac troponin I phospho species in heart areas

1 reference

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7 January 2021

A non-equilibrium isoelectric focusing method to determine states of phosphorylation of cardiac troponin I: identification of Ser-23 and Ser-24 as significant sites of phosphorylation by protein kinase C

1 reference

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7 January 2021

Troponin T: genetics, properties and function

1 reference

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7 January 2021

Global quantitative phosphoprotein analysis using Multiplexed Proteomics technology

1 reference

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Alterations in thin filament regulation induced by a human cardiac troponin T mutant that causes dilated cardiomyopathy are distinct from those induced by troponin T mutants that cause hypertrophic cardiomyopathy

1 reference

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7 January 2021

In vitro motility analysis of actin-tropomyosin regulation by troponin and calcium. The thin filament is switched as a single cooperative unit

1 reference

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7 January 2021

Troponin I and troponin T interact with troponin C to produce different Ca2+-dependent effects on actin-tropomyosin filament motility

1 reference

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A simple method for automatic tracking of actin filaments in the motility assay.

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7 January 2021

Protein kinase C and A sites on troponin I regulate myofilament Ca2+ sensitivity and ATPase activity in the mouse myocardium

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Protein phosphorylation in isolated trabeculae from nonfailing and failing human hearts

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7 January 2021

Beta-protein kinase C and hypertrophic signaling in human heart failure

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7 January 2021

Thin-filament-based modulation of contractile performance in human heart failure

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7 January 2021

Molecular mechanisms of reduced sarcoplasmic reticulum Ca(2+) uptake in human failing left ventricular myocardium

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Increased expression of cardiac phosphatases in patients with end-stage heart failure

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Type 1 phosphatase, a negative regulator of cardiac function

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PKC-alpha regulates cardiac contractility and propensity toward heart failure

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Antiadrenergic effects of adenosine A(1) receptor-mediated protein phosphatase 2a activation in the heart

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Sites phosphorylated in bovine cardiac troponin T and I. Characterization by 31P-NMR spectroscopy and phosphorylation by protein kinases

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Phosphorylation specificities of protein kinase C isozymes for bovine cardiac troponin I and troponin T and sites within these proteins and regulation of myofilament properties

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Identification of a functionally critical protein kinase C phosphorylation residue of cardiac troponin T

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Functional analysis of human cardiac troponin by the in vitro motility assay: comparison of adult, foetal and failing hearts

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Is depressed myocyte contractility centrally involved in heart failure?

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Myosin from failing and non-failing human ventricles exhibit similar contractile properties

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7 January 2021

Calcium sensitivity of isometric tension is increased in canine experimental heart failure

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Targeting protein phosphatase 1 in heart failure

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Frequency- and afterload-dependent cardiac modulation in vivo by troponin I with constitutively active protein kinase A phosphorylation sites.

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Severe disease expression of cardiac troponin C and T mutations in patients with idiopathic dilated cardiomyopathy

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Mutations in sarcomere protein genes as a cause of dilated cardiomyopathy

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Dilated cardiomyopathy mutations in three thin filament regulatory proteins result in a common functional phenotype

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PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts

1 reference

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Functional properties of failing human ventricular myocytes

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Cardiac troponin T variants produced by aberrant splicing of multiple exons in animals with high instances of dilated cardiomyopathy

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10.1016/J.YJMCC.2006.08.017

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21 December 2019

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Europe PubMed Central

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21 December 2019

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