(Q83883527)

English

Do proteomics analyses provide insights into reduced oxidative stress in the brain of an Alzheimer disease transgenic mouse model with an M631L amyloid precursor protein substitution and thereby the importance of amyloid-beta-resident methionine 35

scientific article published on 06 June 2012

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scholarly article

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Europe PubMed Central

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28 January 2020

Do proteomics analyses provide insights into reduced oxidative stress in the brain of an Alzheimer disease transgenic mouse model with an M631L amyloid precursor protein substitution and thereby the importance of amyloid-beta-resident methionine 35 in Alzheimer disease pathogenesis? (English)

1 reference

Europe PubMed Central

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:22500616%20AND%20SRC:MED&resulttype=core&format=json

28 January 2020

Renã A. S. Robinson

2

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28 January 2020

author name string

Rukhsana Sultana

1

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28 January 2020

Miranda Bader Lange

3

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28 January 2020

Ada Fiorini

4

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28 January 2020

Veronica Galvan

5

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28 January 2020

Joanna Fombonne

6

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28 January 2020

Austin Baker

7

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28 January 2020

Olivia Gorostiza

8

1 reference

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28 January 2020

Junli Zhang

9

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28 January 2020

Jian Cai

10

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28 January 2020

William M Pierce

11

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28 January 2020

Dale E Bredesen

12

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28 January 2020

D Allan Butterfield

13

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28 January 2020

publication date

6 June 2012

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28 January 2020

Antioxidants & Redox Signaling

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28 January 2020

17

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28 January 2020

11

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28 January 2020

1507-1514

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28 January 2020

In vivo oxidative stress in brain of Alzheimer disease transgenic mice: Requirement for methionine 35 in amyloid beta-peptide of APP.

1 reference

https://pubmed.ncbi.nlm.nih.gov/22500616

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Differential expression and redox proteomics analyses of an Alzheimer disease transgenic mouse model: effects of the amyloid-β peptide of amyloid precursor protein

1 reference

https://pubmed.ncbi.nlm.nih.gov/22500616

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

In vitro and in vivo oxidative stress associated with Alzheimer's amyloid beta-peptide (1-42)

1 reference

https://pubmed.ncbi.nlm.nih.gov/22500616

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Oxidative stress precedes fibrillar deposition of Alzheimer's disease amyloid beta-peptide (1-42) in a transgenic Caenorhabditis elegans model.

1 reference

https://pubmed.ncbi.nlm.nih.gov/22500616

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Roles of amyloid beta-peptide-associated oxidative stress and brain protein modifications in the pathogenesis of Alzheimer's disease and mild cognitive impairment

1 reference

https://pubmed.ncbi.nlm.nih.gov/22500616

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Estimation of the Genetic Contribution of Presenilin-1 and -2 Mutations in a Population-Based Study of Presenile Alzheimer Disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/22500616

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identification of eukaryotic parvulin homologues: a new subfamily of peptidylprolyl cis-trans isomerases

1 reference

https://pubmed.ncbi.nlm.nih.gov/22500616

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The prolyl isomerase Pin1 restores the function of Alzheimer-associated phosphorylated tau protein

1 reference

https://pubmed.ncbi.nlm.nih.gov/22500616

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Amyloid beta-protein and the genetics of Alzheimer's disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/22500616

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1089/ARS.2011.4470

1 reference

Europe PubMed Central

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28 January 2020

1 reference

Europe PubMed Central

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28 January 2020

1 reference

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28 January 2020

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