(Q24198013)

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Hypothalamic-pituitary-adrenal (HPA) axis suppression after treatment with glucocorticoid therapy for childhood acute lymphoblastic leukaemia

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Hypothalamic-pituitary-adrenal (HPA) axis suppression after treatment with glucocorticoid therapy for childhood acute lymphoblastic leukaemia (English)

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Gertjan Kaspers

5

object named as

Gertjan JL Kaspers

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author name string

Maartje S Gordijn

1

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Reinoud JBJ Gemke

2

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Elvira C van Dalen

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Joost Rotteveel

4

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publication date

16 May 2012

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Cochrane Database of Systematic Reviews

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Early adrenocortical recovery after glucocorticoid therapy in children with leukemia

1 reference

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Adrenal axis function after high-dose steroid therapy for childhood acute lymphoblastic leukemia

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Time course of recovery of adrenal function in children treated for leukemia

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Evaluation of the hypothalamic-pituitary-adrenal axis in children with leukemia before and after 6 weeks of high-dose glucocorticoid therapy

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Impaired adrenal function after glucocorticoid therapy in children with acute lymphoblastic leukemia

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Clinical impact of corticosteroid-induced adrenal suppression during treatment for acute lymphoblastic leukemia in children: a prospective observational study using the low-dose adrenocorticotropin test

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Glucocorticoids in the treatment of children with acute lymphoblastic leukemia and hodgkin's disease: a pilot study on the adverse psychological reactions and possible associations with neurobiological, endocrine, and genetic markers.

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Tests of adrenal insufficiency

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Possible association between high-dose fluconazole and adrenal insufficiency in critically ill patients

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Comparison of adrenal function tests in children--the glucagon stimulation test allows the simultaneous assessment of adrenal function and growth hormone response in children

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Cohort studies: marching towards outcomes

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Cytokine-stimulated human dermal microvascular endothelial cells produce interleukin 6--inhibition by hydrocortisone, dexamethasone, and calcitriol.

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No advantage of dexamethasone over prednisolone for the outcome of standard- and intermediate-risk childhood acute lymphoblastic leukemia in the Tokyo Children's Cancer Study Group L95-14 protocol

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Glucocorticoid-induced adrenal insufficiency

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Evaluation of adrenal function in patients with growth hormone deficiency and hypothalamic-pituitary disorders: comparison between insulin-induced hypoglycemia, low-dose ACTH, standard ACTH and CRH stimulation tests

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Glucocorticoid-induced apoptosis in lymphocytes

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Induction death and treatment-related mortality in first remission of children with acute lymphoblastic leukemia: a population-based analysis of the Austrian Berlin-Frankfurt-Münster study group.

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Hazards of pharmacological tests of growth hormone secretion in childhood

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Acute adrenal failure associated with fluconazole after administration of high-dose cyclophosphamide

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Adrenal insufficiency: still a cause of morbidity and death in childhood

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Dexamethasone in the maintenance phase of acute lymphoblastic leukaemia treatment: is the risk of lethal infections too high?

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Role of corticotropin-releasing hormone testing in assessment of hypothalamic-pituitary-adrenal axis function in infants with congenital central hypothyroidism

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The glucagon stimulation test: effect of plasma growth hormone and on immunoreactive insulin, cortisol, and glucose in children.

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Treatment related deaths during induction and in first remission in acute lymphoblastic leukaemia: MRC UKALL X.

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Death during induction therapy and first remission of acute leukemia in childhood: the St. Jude experience

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Growth and endocrinological disorders up to 21 years after treatment for acute lymphoblastic leukemia in childhood

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Corticosteroids and interferons inhibit cytokine-induced production of IL-8 by human endothelial cells

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Intramuscular glucagon as a provocative stimulus for the assessment of pituitary function: growth hormone and cortisol responses

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Suppression and recovery of adrenal response after short-term, high-dose glucocorticoid treatment

1 reference

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Low-dose (1 microgram) adrenocorticotrophin (ACTH) stimulation as a screening test for impaired hypothalamo-pituitary-adrenal axis function: sensitivity, specificity and accuracy in comparison with the high-dose (250 microgram) test

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The Effect of Long-Term Glucocorticoid Therapy on Pituitary–Adrenal Responses to Exogenous Corticotropin-Releasing Hormone

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Treatment-related death in childhood acute lymphoblastic leukaemia in the Nordic countries: 1992-2001

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based on heuristic

inferred from DOI database lookup

One microgram is the lowest ACTH dose to cause a maximal cortisol response. There is no diurnal variation of cortisol response to submaximal ACTH stimulation

1 reference

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7 January 2021

based on heuristic

inferred from DOI database lookup

[Evaluation of hypothalamic-pituitary-adrenal axis recovery after corticotherapy by using basal cortisol secretion]

1 reference

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7 January 2021

based on heuristic

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10.1002/14651858.CD008727.PUB2

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