(Q24300759)

negative regulation of sodium ion transmembrane transport

GOA release 2020-03-11

1 reference

Journal of Biological Chemistry

GOA release 2020-03-11

4

Fiona J McDonald

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author name string

Wolfgang Biasio

series ordinal

1

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Tina Chang

series ordinal

2

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C Joy McIntosh

series ordinal

3

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language of work or name

English

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publication date

13 February 2004

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published in

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volume

279

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issue

7

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page(s)

5429-34

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cites work

Amiloride-sensitive epithelial Na+ channel is made of three homologous subunits

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Mutations in subunits of the epithelial sodium channel cause salt wasting with hyperkalaemic acidosis, pseudohypoaldosteronism type 1

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Hypertension caused by a truncated epithelial sodium channel gamma subunit: genetic heterogeneity of Liddle syndrome

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A de novo missense mutation of the beta subunit of the epithelial sodium channel causes hypertension and Liddle syndrome, identifying a proline-rich segment critical for regulation of channel activity

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A mutation in the epithelial sodium channel causing Liddle disease increases channel activity in the Xenopus laevis oocyte expression system

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Liddle's syndrome: heritable human hypertension caused by mutations in the beta subunit of the epithelial sodium channel

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Mechanism by which Liddle's syndrome mutations increase activity of a human epithelial Na+ channel

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Molecular cloning and functional expression of a novel amiloride-sensitive Na+ channel

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A new subunit of the epithelial Na+ channel identifies regions involved in Na+ self-inhibition

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Rabbit retinal neurons and glia express a variety of ENaC/DEG subunits

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Protons at the gate: DEG/ENaC ion channels help us feel and remember

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Inhibition of the epithelial Na+ channel by interaction of Nedd4 with a PY motif deleted in Liddle's syndrome.

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Defective regulation of the epithelial Na+ channel by Nedd4 in Liddle's syndrome.

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From Src Homology domains to other signaling modules: proposal of the 'protein recognition code'.

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Identification of a new copper metabolism gene by positional cloning in a purebred dog population

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Interactions between subunits of the human epithelial sodium channel.

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Recombination-mediated PCR-directed plasmid construction in vivo in yeast

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Mouse U2af1-rs1 is a neomorphic imprinted gene

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The molecular basis of copper-transport diseases

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The copper toxicosis gene product Murr1 directly interacts with the Wilson disease protein

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Regulation of the amiloride-sensitive epithelial sodium channel by syntaxin 1A.

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Interaction of syntaxins with the amiloride-sensitive epithelial sodium channel

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Syntaxin 1A regulates ENaC via domain-specific interactions.

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Copper-induced apical trafficking of ATP7B in polarized hepatoma cells provides a mechanism for biliary copper excretion

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Hepatic lysosomal copper protein in dogs with an inherited copper toxicosis

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inferred from DOI database lookup

7 January 2021

based on heuristic

Cloning and expression of the beta- and gamma-subunits of the human epithelial sodium channel

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M311155200