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TNF-alpha induces two distinct caspase-8 activation pathways
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instance of
scholarly article
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title
TNF-alpha induces two distinct caspase-8 activation pathways
(English)
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main subject
TNF
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author name string
Lai Wang
series ordinal
1
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Fenghe Du
series ordinal
2
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Xiaodong Wang
series ordinal
3
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language of work or name
English
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publication date
16 May 2008
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published in
Cell
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volume
133
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issue
4
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page(s)
693-703
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cites work
RIP: a novel protein containing a death domain that interacts with Fas/APO-1 (CD95) in yeast and causes cell death
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Autocrine TNFalpha signaling renders human cancer cells susceptible to Smac-mimetic-induced apoptosis
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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The E3 ubiquitin ligase itch couples JNK activation to TNFalpha-induced cell death by inducing c-FLIP(L) turnover
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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IAP antagonists induce autoubiquitination of c-IAPs, NF-kappaB activation, and TNFalpha-dependent apoptosis
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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IAP antagonists target cIAP1 to induce TNFalpha-dependent apoptosis
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Ubiquitination of RIP1 regulates an NF-kappaB-independent cell-death switch in TNF signaling
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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Regulation of early wave of germ cell apoptosis and spermatogenesis by deubiquitinating enzyme CYLD.
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Life and death decisions: secondary complexes and lipid rafts in TNF receptor family signal transduction
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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Activation of IKK by TNFalpha requires site-specific ubiquitination of RIP1 and polyubiquitin binding by NEMO
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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A JNK-Dependent Pathway Is Required for TNFα-Induced Apoptosis
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Tumor necrosis factor: an apoptosis JuNKie?
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TNF-dependent recruitment of the protein kinase RIP to the TNF receptor-1 signaling complex
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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The death domain kinase RIP mediates the TNF-induced NF-kappaB signal
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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Structural and biochemical basis of apoptotic activation by Smac/DIABLO
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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Structural basis for binding of Smac/DIABLO to the XIAP BIR3 domain
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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7 January 2021
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Structural basis of IAP recognition by Smac/DIABLO
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reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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Inhibition of death receptor signals by cellular FLIP
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Crossref
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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7 January 2021
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Identification of the familial cylindromatosis tumour-suppressor gene
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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7 January 2021
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Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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Nuclear factor-kappaB in cancer development and progression
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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7 January 2021
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Sensing of Lys 63-linked polyubiquitination by NEMO is a key event in NF-kappaB activation [corrected]
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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7 January 2021
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Lasker Clinical Medical Research Award. TNF defined as a therapeutic target for rheumatoid arthritis and other autoimmune diseases
1 reference
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reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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7 January 2021
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Suppression of tumor necrosis factor-induced cell death by inhibitor of apoptosis c-IAP2 is under NF-kappaB control
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Crossref
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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7 January 2021
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inferred from DOI database lookup
An APAF-1.cytochrome c multimeric complex is a functional apoptosome that activates procaspase-9
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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7 January 2021
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Phosphatidylinositol 3-kinase/Akt activity regulates c-FLIP expression in tumor cells
1 reference
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https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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7 January 2021
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inferred from DOI database lookup
Smac/DIABLO selectively reduces the levels of c-IAP1 and c-IAP2 but not that of XIAP and livin in HeLa cells
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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7 January 2021
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inferred from DOI database lookup
Ubiquitination of RIP Is Required for Tumor Necrosis Factor α-induced NF-κB Activation
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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7 January 2021
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TNF-R1 signaling: a beautiful pathway
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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7 January 2021
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inferred from DOI database lookup
The PIDDosome, a protein complex implicated in activation of caspase-2 in response to genotoxic stress
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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7 January 2021
based on heuristic
inferred from DOI database lookup
A small molecule Smac mimic potentiates TRAIL- and TNFalpha-mediated cell death
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
NF-kappaB antiapoptosis: induction of TRAF1 and TRAF2 and c-IAP1 and c-IAP2 to suppress caspase-8 activation
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Ubiquitin protein ligase activity of IAPs and their degradation in proteasomes in response to apoptotic stimuli
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Distinct signaling pathways in TRAIL- versus tumor necrosis factor-induced apoptosis
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
NF-kappaB inducers upregulate cFLIP, a cycloheximide-sensitive inhibitor of death receptor signaling
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
NF-kappaB signals induce the expression of c-FLIP.
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Posttranscriptional downregulation of c-IAP2 by the ubiquitin protein ligase c-IAP1 in vivo
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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7 January 2021
based on heuristic
inferred from DOI database lookup
Competitive control of independent programs of tumor necrosis factor receptor-induced cell death by TRADD and RIP1
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FJ.CELL.2008.03.036
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7 January 2021
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Identifiers
DOI
10.1016/J.CELL.2008.03.036
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
465500
OpenCitations bibliographic resource ID
465500
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
465500
PubMed publication ID
18485876
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
465500
ResearchGate publication ID
5364426
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