(Q24655350)

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Elimination of KATP channels in mouse islets results in elevated [U-13C]glucose metabolism, glutaminolysis, and pyruvate cycling but a decreased gamma-aminobutyric acid shunt

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Elimination of KATP channels in mouse islets results in elevated [U-13C]glucose metabolism, glutaminolysis, and pyruvate cycling but a decreased gamma-aminobutyric acid shunt (English)

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Charles A Stanley

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Charles A Stanley

10

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author name string

Changhong Li

1

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Itzhak Nissim

2

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Pan Chen

3

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Carol Buettger

4

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Habiba Najafi

5

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Yevgeny Daikhin

6

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Ilana Nissim

7

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Heather W Collins

8

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Marc Yudkoff

9

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Franz M Matschinsky

11

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language of work or name

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publication date

20 June 2008

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Journal of Biological Chemistry

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283

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25

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17238-49

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13C NMR isotopomer analysis reveals a connection between pyruvate cycling and glucose-stimulated insulin secretion (GSIS)

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7 April 2017

A role for ATP-citrate lyase, malic enzyme, and pyruvate/citrate cycling in glucose-induced insulin secretion

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7 April 2017

Metabolic and ionic coupling factors in amino acid-stimulated insulin release in pancreatic beta-HC9 cells.

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28 September 2017

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28 September 2017

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28 September 2017

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Sur1 knockout mice. A model for K(ATP) channel-independent regulation of insulin secretion

1 reference

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Effects of glucose on insulin release and on intermediary metabolism of isolated perifused pancreatic islets from fed and fasted rats.

1 reference

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1 reference

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1 reference

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10.1074/JBC.M709235200

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