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The role of ALFY in selective autophagy
scientific article
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scholarly article
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PubMed
title
The role of ALFY in selective autophagy
(English)
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stated in
PubMed
main subject
autophagy
0 references
author name string
P Isakson
series ordinal
1
1 reference
stated in
Crossref
P Holland
series ordinal
2
1 reference
stated in
Crossref
A Simonsen
series ordinal
3
1 reference
stated in
Crossref
language of work or name
English
1 reference
stated in
PubMed
publication date
January 2013
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stated in
PubMed
published in
Cell Death & Differentiation
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stated in
PubMed
volume
20
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stated in
PubMed
page(s)
12-20
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stated in
PubMed
issue
1
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PubMed
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The selective macroautophagic degradation of aggregated proteins requires the PI3P-binding protein Alfy
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p62/SQSTM1 and ALFY interact to facilitate the formation of p62 bodies/ALIS and their degradation by autophagy
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Structural basis of target recognition by Atg8/LC3 during selective autophagy
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A ubiquitin-like system mediates protein lipidation
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Selective autophagy mediated by autophagic adapter proteins
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p62/SQSTM1 forms protein aggregates degraded by autophagy and has a protective effect on huntingtin-induced cell death
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ALIS are stress-induced protein storage compartments for substrates of the proteasome and autophagy
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28 September 2017
Alfy-dependent elimination of aggregated proteins by macroautophagy: can there be too much of a good thing?
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3524637
retrieved
31 May 2018
Autophagy-related protein 8 (Atg8) family interacting motif in Atg3 mediates the Atg3-Atg8 interaction and is crucial for the cytoplasm-to-vacuole targeting pathway
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3524637
retrieved
31 May 2018
Sequestosome 1/p62 links familial ALS mutant SOD1 to LC3 via an ubiquitin-independent mechanism.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3524637
retrieved
31 May 2018
How shall I eat thee?
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FCDD.2012.66
retrieved
21 January 2018
Aggregate formation inhibits proteasomal degradation of polyglutamine proteins.
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FCDD.2012.66
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21 January 2018
Nucleocytoplasmic shuttling of p62/SQSTM1 and its role in recruitment of nuclear polyubiquitinated proteins to promyelocytic leukemia bodies.
1 reference
stated in
Crossref
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https://api.crossref.org/works/10.1038%2FCDD.2012.66
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21 January 2018
Early and reversible neuropathology induced by tetracycline-regulated lentiviral overexpression of mutant huntingtin in rat striatum
1 reference
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Crossref
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21 January 2018
RNAi suppresses polyglutamine-induced neurodegeneration in a model of spinocerebellar ataxia.
1 reference
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Crossref
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21 January 2018
Recovery from polyglutamine-induced neurodegeneration in conditional SCA1 transgenic mice.
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1038%2FCDD.2012.66
retrieved
21 January 2018
Atg11 Directs Autophagosome Cargoes to the PAS Along Actin Cables
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FCDD.2012.66
retrieved
21 January 2018
A Reassessment of the Neuropathology of Frontotemporal Dementia Linked to Chromosome 3
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/22653340
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Shigella phagocytic vacuolar membrane remnants participate in the cellular response to pathogen invasion and are regulated by autophagy
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/22653340
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1038/CDD.2012.66
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
2337416
Dimensions Publication ID
1042606948
0 references
OpenCitations bibliographic resource ID
2337416
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
2337416
PMCID
3524637
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
2337416
PubMed ID
22653340
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
2337416
ResearchGate publication ID
225086206
0 references
Springer Nature article ID
10.1038/cdd.2012.66
0 references
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