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Typhoid fever: "you can't hit what you can't see"
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PubMed
title
Typhoid fever: "you can't hit what you can't see"
(English)
1 reference
stated in
PubMed
main subject
infectious disease
0 references
typhoid fever
1 reference
based on heuristic
inferred from title
author
Andreas J. Bäumler
series ordinal
3
object named as
Andreas J. Bäumler
1 reference
stated in
Crossref
author name string
Tamding Wangdi
series ordinal
1
1 reference
stated in
Crossref
Sebastian E. Winter
series ordinal
2
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stated in
Crossref
language of work or name
English
1 reference
stated in
PubMed
publication date
2012
1 reference
stated in
PubMed
published in
Gut microbes
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stated in
PubMed
volume
3
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stated in
PubMed
issue
2
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stated in
PubMed
page(s)
88-92
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PubMed
cites work
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The Vi capsular polysaccharide prevents complement receptor 3-mediated clearance of Salmonella enterica serotype Typhi
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Intracellular survival of Salmonella enterica serovar Typhi in human macrophages is independent of Salmonella pathogenicity island (SPI)-2.
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A rapid change in virulence gene expression during the transition from the intestinal lumen into tissue promotes systemic dissemination of Salmonella
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A HISTOLOGICAL STUDY OF TYPHOID FEVER.
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The TviA auxiliary protein renders the Salmonella enterica serotype Typhi RcsB regulon responsive to changes in osmolarity
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The capsule-encoding viaB locus reduces intestinal inflammation by a Salmonella pathogenicity island 1-independent mechanism
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Coordinated regulation of expression of Salmonella pathogenicity island 1 and flagellar type III secretion systems by ATP-dependent ClpXP protease
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The Vi-capsule prevents Toll-like receptor 4 recognition of Salmonella.
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The Salmonella enterica serotype Typhi regulator TviA reduces interleukin-8 production in intestinal epithelial cells by repressing flagellin secretion
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Outbreaks of typhoid fever in the United States, 1960-99.
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Salmonella typhi flagella are potent inducers of proinflammatory cytokine secretion by human monocytes.
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Environmental regulation of Salmonella typhi invasion-defective mutants
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Survival of Vi-capsulated and Vi-deleted Salmonella typhi strains in cultured macrophage expressing different levels of CD14 antigen
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Characterization of the rcsA and rcsB genes from Salmonella typhi: rcsB through tviA is involved in regulation of Vi antigen synthesis
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Characterization of defined ompR mutants of Salmonella typhi: ompR is involved in the regulation of Vi polysaccharide expression
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Histopathology of the upper small intestines in typhoid fever. Biopsy study of experimental disease in man.
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Genetic Transfer of the Vi Antigen from Salmonella typhosa to Escherichia coli
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Cloning and molecular characterization of genes whose products allow Salmonella typhimurium to penetrate tissue culture cells
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The opsonizing ligand on Salmonella typhimurium influences incorporation of specific, but not azurophil, granule constituents into neutrophil phagosomes
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Transcriptional analysis of the flagellar regulon of Salmonella typhimurium
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Inhibition of chemotaxis of neutrophil leukocytes to interleukin-8 by endotoxins of various bacteria
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Incubation period, severity of disease, and infecting dose: evidence from a Salmonella outbreak
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Identification of a Salmonella typhimurium invasion locus by selection for hyperinvasive mutants
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The RcsB-RcsC regulatory system of Salmonella typhi differentially modulates the expression of invasion proteins, flagellin and Vi antigen in response to osmolarity.
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inferred from PubMed ID database lookup
Identifiers
DOI
10.4161/GMIC.18602
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4823219
OpenCitations bibliographic resource ID
4823219
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4823219
PMCID
3370952
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4823219
PubMed ID
22156762
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4823219
ResearchGate publication ID
51866910
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