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The GSK-3 family as therapeutic target for myocardial diseases
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PubMed
review article
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Europe PubMed Central
title
The GSK-3 family as therapeutic target for myocardial diseases
(English)
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PubMed
author
Hind Lal
object named as
H. Lal
series ordinal
1
0 references
Thomas Force
object named as
T. Force
series ordinal
4
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James R. Woodgett
object named as
J. Woodgett
series ordinal
3
0 references
Firdos Ahmad
object named as
F. Ahmad
series ordinal
2
0 references
language of work or name
English
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stated in
PubMed
publication date
2 January 2015
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stated in
PubMed
published in
Circulation Research
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stated in
PubMed
volume
116
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stated in
PubMed
issue
1
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stated in
PubMed
page(s)
138-49
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stated in
PubMed
cites work
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Glycogen synthase kinase-3beta regulates post-myocardial infarction remodeling and stress-induced cardiomyocyte proliferation in vivo
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Constitutive glycogen synthase kinase-3alpha/beta activity protects against chronic beta-adrenergic remodelling of the heart.
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28 September 2017
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28 September 2017
Deletion of GSK-3beta in mice leads to hypertrophic cardiomyopathy secondary to cardiomyoblast hyperproliferation
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28 September 2017
GSK-3beta in mouse fibroblasts controls wound healing and fibrosis through an endothelin-1-dependent mechanism
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28 September 2017
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28 September 2017
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28 September 2017
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28 September 2017
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28 September 2017
Glycogen synthase kinase 3 (GSK3) in the heart: a point of integration in hypertrophic signalling and a therapeutic target? A critical analysis
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28 September 2017
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28 September 2017
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28 September 2017
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28 September 2017
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28 September 2017
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28 September 2017
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28 September 2017
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28 September 2017
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31 May 2018
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31 May 2018
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28 November 2018
Differential roles of GSK-3β during myocardial ischemia and ischemia/reperfusion.
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28 November 2018
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28 November 2018
Glycogen synthase kinase-3 inactivation is not required for ischemic preconditioning or postconditioning in the mouse
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28 November 2018
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28 November 2018
Inhibition of glycogen synthase kinase 3beta during heart failure is protective
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28 November 2018
Glycogen synthase kinase-3beta regulates growth, calcium homeostasis, and diastolic function in the heart
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4283216
retrieved
28 November 2018
Phosphorylation of glycogen synthase kinase-3beta during preconditioning through a phosphatidylinositol-3-kinase--dependent pathway is cardioprotective
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4283216
retrieved
28 November 2018
Glycogen synthase kinase-3alpha reduces cardiac growth and pressure overload-induced cardiac hypertrophy by inhibition of extracellular signal-regulated kinases
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/25552693
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Predominance of beta-catenin mutations and beta-catenin dysregulation in sporadic aggressive fibromatosis (desmoid tumor)
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/25552693
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
The Akt-glycogen synthase kinase 3beta pathway regulates transcription of atrial natriuretic factor induced by beta-adrenergic receptor stimulation in cardiac myocytes
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/25552693
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Modulation of the mitochondrial permeability transition pore complex in GSK-3beta-mediated myocardial protection
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/25552693
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1161/CIRCRESAHA.116.303613
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4008698
Fatcat ID
release_ywshgodsh5fktbcp7bsc7wedte
0 references
OpenCitations bibliographic resource ID
4008698
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4008698
PMC publication ID
4283216
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4008698
PubMed publication ID
25552693
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4008698
ResearchGate publication ID
271587549
0 references
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