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Pernicious attrition and inter-RyR2 CICR current control in cardiac muscle
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title
Pernicious attrition and inter-RyR2 CICR current control in cardiac muscle
(English)
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author name string
Dirk Gillespie
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1
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Michael Fill
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2
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language of work or name
English
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publication date
May 2013
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published in
Journal of Molecular and Cellular Cardiology
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volume
58
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page(s)
53-8
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cites work
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How does stochastic ryanodine receptor-mediated Ca leak fail to initiate a Ca spark?
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Unitary Ca2+ current through mammalian cardiac and amphibian skeletal muscle ryanodine receptor Channels under near-physiological ionic conditions
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Ryanodine receptor current amplitude controls Ca2+ sparks in cardiac muscle
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(De)constructing the ryanodine receptor: modeling ion permeation and selectivity of the calcium release channel.
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Regulation of the gating of the sheep cardiac sarcoplasmic reticulum Ca(2+)-release channel by luminal Ca2+.
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Electron probe X-ray microanalysis of post-tetanic Ca2+ and Mg2+ movements across the sarcoplasmic reticulum in situ
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Modelling cardiac calcium sparks in a three-dimensional reconstruction of a calcium release unit
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Identification of changes in the functional profile of the cardiac ryanodine receptor caused by the coupled gating phenomenon
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Sarcoplasmic reticulum calcium content fluctuation is the key to cardiac alternans
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Ca2+ scraps: local depletions of free [Ca2+] in cardiac sarcoplasmic reticulum during contractions leave substantial Ca2+ reserve
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Coupled gating between cardiac calcium release channels (ryanodine receptors).
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28 November 2018
Numerical simulation of local calcium movements during L-type calcium channel gating in the cardiac diad
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Ca2+ 'sparks' and waves in intact ventricular muscle resolved by confocal imaging
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Ca2+ sparks and Ca2+ waves in saponin-permeabilized rat ventricular myocytes
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Arrhythmogenesis in catecholaminergic polymorphic ventricular tachycardia: insights from a RyR2 R4496C knock-in mouse model
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Termination of calcium-induced calcium release by induction decay: an emergent property of stochastic channel gating and molecular scale architecture
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RyR2 mutations linked to ventricular tachycardia and sudden death reduce the threshold for store-overload-induced Ca2+ release (SOICR)
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Alteration of sarcoplasmic reticulum Ca2+ release termination by ryanodine receptor sensitization and in heart failure.
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Luminal Ca2+ controls termination and refractory behavior of Ca2+-induced Ca2+ release in cardiac myocytes
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10.1016/J.YJMCC.2013.01.011
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PMC publication ID
3628281
1 reference
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PubMed
PubMed publication ID
23369697
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PubMed
ResearchGate publication ID
235390555
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