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Epithelial-Mesenchymal Plasticity: A Central Regulator of Cancer Progression
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PubMed
title
Epithelial-Mesenchymal Plasticity: A Central Regulator of Cancer Progression
(English)
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stated in
PubMed
author name string
Xin Ye
series ordinal
1
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stated in
Crossref
Robert A. Weinberg
series ordinal
2
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stated in
Crossref
language of work or name
English
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stated in
PubMed
publication date
November 2015
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stated in
PubMed
published in
Trends in Cell Biology
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stated in
PubMed
volume
25
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PubMed
issue
11
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PubMed
page(s)
675-86
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PubMed
cites work
An EMT-driven alternative splicing program occurs in human breast cancer and modulates cellular phenotype
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The ZEB/miR-200 feedback loop--a motor of cellular plasticity in development and cancer?
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A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells
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The transcriptional repressor Snail promotes mammary tumor recurrence
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Residual breast cancers after conventional therapy display mesenchymal as well as tumor-initiating features
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Stem-cell-like properties and epithelial plasticity arise as stable traits after transient Twist1 activation
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A breast cancer stem cell niche supported by juxtacrine signalling from monocytes and macrophages
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Dihydropyrimidine accumulation is required for the epithelial-mesenchymal transition
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Transient SNAIL1 expression is necessary for metastatic competence in breast cancer
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A signature of epithelial-mesenchymal plasticity and stromal activation in primary tumor modulates late recurrence in breast cancer independent of disease subtype.
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Targeting WNT1-inducible signaling pathway protein 2 alters human breast cancer cell susceptibility to specific lysis through regulation of KLF-4 and miR-7 expression.
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A positive feedback loop between mesenchymal-like cancer cells and macrophages is essential to breast cancer metastasis
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Epithelial mesenchymal transition in early invasive breast cancer: an immunohistochemical and reverse phase protein array study
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TGF-β-Id1 signaling opposes Twist1 and promotes metastatic colonization via a mesenchymal-to-epithelial transition
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The epigenetics of epithelial-mesenchymal plasticity in cancer
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Epithelial plasticity: a common theme in embryonic and cancer cells.
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Epigenetic regulation of EMT: the Snail story
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Poised chromatin at the ZEB1 promoter enables breast cancer cell plasticity and enhances tumorigenicity
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The collagen receptor discoidin domain receptor 2 stabilizes SNAIL1 to facilitate breast cancer metastasis
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Epithelial-to-mesenchymal transition and autophagy induction in breast carcinoma promote escape from T-cell-mediated lysis.
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Slug controls stem/progenitor cell growth dynamics during mammary gland morphogenesis
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Metastatic colonization requires the repression of the epithelial-mesenchymal transition inducer Prrx1.
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Epithelial-to-mesenchymal transition (EMT) confers primary resistance to trastuzumab (Herceptin).
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29 September 2017
Cancer-stimulated mesenchymal stem cells create a carcinoma stem cell niche via prostaglandin E2 signaling
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ZEB/miR-200 feedback loop: at the crossroads of signal transduction in cancer
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EMT inducers catalyze malignant transformation of mammary epithelial cells and drive tumorigenesis towards claudin-low tumors in transgenic mice.
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Slug and Sox9 cooperatively determine the mammary stem cell state
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Interactions between cancer stem cells and their niche govern metastatic colonization
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A p53/miRNA-34 axis regulates Snail1-dependent cancer cell epithelial-mesenchymal transition.
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Multistep nature of metastatic inefficiency: dormancy of solitary cells after successful extravasation and limited survival of early micrometastases.
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27 November 2018
MiR-34 and SNAIL: another double-negative feedback loop controlling cellular plasticity/EMT governed by p53.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4628843
retrieved
27 November 2018
Epithelial-mesenchymal transition and stemness features in circulating tumor cells from breast cancer patients.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4628843
retrieved
27 November 2018
Parallel progression of primary tumours and metastases
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4628843
retrieved
27 November 2018
Diverse cellular and molecular mechanisms contribute to epithelial plasticity and metastasis.
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4628843
retrieved
27 November 2018
TGFbeta signaling is necessary for carcinoma cell invasiveness and metastasis
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4628843
retrieved
27 November 2018
Temporal progression of metastasis in lung: cell survival, dormancy, and location dependence of metastatic inefficiency
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/26437589
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1016/J.TCB.2015.07.012
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
892022
Fatcat ID
release_43jotemxwbapxh4umjsujktqse
0 references
OpenCitations bibliographic resource ID
892022
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
892022
PMC publication ID
4628843
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
892022
PubMed publication ID
26437589
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
892022
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