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The permissive role of glucocorticoids in neuroinflammatory priming: mechanisms and insights
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scholarly article
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PubMed
review article
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Europe PubMed Central
title
The permissive role of glucocorticoids in neuroinflammatory priming: mechanisms and insights
(English)
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stated in
PubMed
main subject
human nutrition
0 references
glucocorticoid
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author
Linda R. Watkins
series ordinal
2
object named as
Linda R. Watkins
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Crossref
author name string
Matthew G. Frank
series ordinal
1
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Crossref
Steven F. Maier
series ordinal
3
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Crossref
language of work or name
English
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PubMed
publication date
August 2015
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stated in
PubMed
published in
Current Opinion in Endocrinology, Diabetes and Obesity
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stated in
PubMed
volume
22
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PubMed
issue
4
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stated in
PubMed
page(s)
300-5
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PubMed
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Chronic exposure to exogenous glucocorticoids primes microglia to pro-inflammatory stimuli and induces NLRP3 mRNA in the hippocampus
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Stress induces the danger-associated molecular pattern HMGB-1 in the hippocampus of male Sprague Dawley rats: a priming stimulus of microglia and the NLRP3 inflammasome.
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The HPA - Immune Axis and the Immunomodulatory Actions of Glucocorticoids in the Brain
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The five Rs of glucocorticoid action during inflammation: ready, reinforce, repress, resolve, and restore.
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Glucocorticoids sensitize the innate immune system through regulation of the NLRP3 inflammasome.
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The stressed CNS: when glucocorticoids aggravate inflammation.
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29 September 2017
Signaling to NF-kappaB by Toll-like receptors
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29 September 2017
The influence of systemic inflammation on inflammation in the brain: implications for chronic neurodegenerative disease
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29 September 2017
Chronic stress enhances microglia activation and exacerbates death of nigral dopaminergic neurons under conditions of inflammation.
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2 June 2018
Glucocorticoids mediate stress-induced priming of microglial pro-inflammatory responses
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2 June 2018
Stress increases vulnerability to inflammation in the rat prefrontal cortex.
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retrieved
27 November 2018
Identifiers
DOI
10.1097/MED.0000000000000168
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4854340
OpenCitations bibliographic resource ID
4854340
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4854340
PMC publication ID
4516217
1 reference
stated in
PubMed
PubMed publication ID
26087336
1 reference
stated in
PubMed
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