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Docetaxel-induced polyploidization may underlie chemoresistance and disease relapse
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instance of
scholarly article
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stated in
PubMed
review article
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stated in
Europe PubMed Central
title
Docetaxel-induced polyploidization may underlie chemoresistance and disease relapse
(English)
1 reference
stated in
PubMed
main subject
whole genome duplication
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based on heuristic
inferred from title
author name string
Angela Ogden
series ordinal
1
1 reference
stated in
Crossref
Padmashree C.G. Rida
series ordinal
2
1 reference
stated in
Crossref
Beatrice S. Knudsen
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3
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stated in
Crossref
Omer Kucuk
series ordinal
4
1 reference
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Crossref
Ritu Aneja
series ordinal
5
1 reference
stated in
Crossref
language of work or name
English
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stated in
PubMed
publication date
28 October 2015
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stated in
PubMed
published in
Cancer Letters
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stated in
PubMed
volume
367
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stated in
PubMed
issue
2
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PubMed
page(s)
89-92
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PubMed
cites work
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Phase III study of doxorubicin/cyclophosphamide with concomitant versus sequential docetaxel as adjuvant treatment in patients with human epidermal growth factor receptor 2-normal, node-positive breast cancer: BCIRG-005 trial
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Secondary leukaemia after cure for locally advanced NSCLC: alkylating type secondary leukaemia after induction therapy with docetaxel and carboplatin for NSCLC IIIB.
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Neosis and its potential role in cancer development and chemoresistance.
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Survival of docetaxel-resistant prostate cancer cells in vitro depends on phenotype alterations and continuity of drug exposure
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P27KiP1 overexpression inhibits the growth and doxorubicin sensitivity of HT29 human colon cancer cells in vivo
1 reference
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reference URL
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retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1016/J.CANLET.2015.06.025
0 references
PMC publication ID
4813805
1 reference
stated in
PubMed
PubMed publication ID
26185000
1 reference
stated in
PubMed
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