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Inositol Polyphosphate-5-Phosphatase F (INPP5F) inhibits STAT3 activity and suppresses gliomas tumorigenicity
scientific journal article
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scholarly article
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PubMed
PubMed ID
25476455
retrieved
4 January 2017
title
Inositol Polyphosphate-5-Phosphatase F (INPP5F) inhibits STAT3 activity and suppresses gliomas tumorigenicity
(English)
1 reference
stated in
PubMed
PubMed ID
25476455
retrieved
4 January 2017
main subject
Inositol polyphosphate-5-phosphatase F
1 reference
stated in
GOA release 2020-03-11
negative regulation of tyrosine phosphorylation of STAT protein
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stated in
GOA release 2020-03-11
author name string
Hong Sug Kim
series ordinal
1
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PubMed
PubMed ID
25476455
retrieved
4 January 2017
Aiguo Li
series ordinal
2
1 reference
stated in
PubMed
PubMed ID
25476455
retrieved
4 January 2017
Susie Ahn
series ordinal
3
1 reference
stated in
PubMed
PubMed ID
25476455
retrieved
4 January 2017
Hua Song
series ordinal
4
1 reference
stated in
PubMed
PubMed ID
25476455
retrieved
4 January 2017
Wei Zhang
series ordinal
5
1 reference
stated in
PubMed
PubMed ID
25476455
retrieved
4 January 2017
language of work or name
English
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publication date
5 December 2014
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stated in
PubMed
PubMed ID
25476455
retrieved
4 January 2017
published in
Scientific Reports
1 reference
stated in
PubMed
PubMed ID
25476455
retrieved
4 January 2017
volume
4
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stated in
PubMed
PubMed ID
25476455
retrieved
4 January 2017
page(s)
7330
1 reference
stated in
PubMed
PubMed ID
25476455
retrieved
4 January 2017
copyright license
Creative Commons Attribution 4.0 International
start time
5 December 2014
1 reference
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April 2022 Public Data File from Crossref
copyright status
copyrighted
0 references
cites work
The coiled-coil domain of Stat3 is essential for its SH2 domain-mediated receptor binding and subsequent activation induced by epidermal growth factor and interleukin-6
1 reference
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PubMed Central
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20 March 2017
Phosphorylation of EZH2 activates STAT3 signaling via STAT3 methylation and promotes tumorigenicity of glioblastoma stem-like cells
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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20 March 2017
The tumor suppressor, PTEN/MMAC1, dephosphorylates the lipid second messenger, phosphatidylinositol 3,4,5-trisphosphate
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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20 March 2017
Comprehensive genomic characterization defines human glioblastoma genes and core pathways
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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20 March 2017
The tyrosine phosphatase PTPRD is a tumor suppressor that is frequently inactivated and mutated in glioblastoma and other human cancers
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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20 March 2017
Identification and characterization of a sac domain-containing phosphoinositide 5-phosphatase
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PubMed Central
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20 March 2017
Specific inhibition of Stat3 signal transduction by PIAS3
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PubMed Central
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20 March 2017
Hdac2 regulates the cardiac hypertrophic response by modulating Gsk3 beta activity
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PubMed Central
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20 March 2017
A Novel Sequence in the Coiled-coil Domain of Stat3 Essential for Its Nuclear Translocation
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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20 March 2017
Inpp5f is a polyphosphoinositide phosphatase that regulates cardiac hypertrophic responsiveness
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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20 March 2017
The evolution of phosphatidylinositol 3-kinases as regulators of growth and metabolism
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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7 April 2017
Stats: transcriptional control and biological impact
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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7 April 2017
STATs and gene regulation
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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7 April 2017
IL-6 and Stat3 are required for survival of intestinal epithelial cells and development of colitis-associated cancer
1 reference
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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7 April 2017
Tumor stem cells derived from glioblastomas cultured in bFGF and EGF more closely mirror the phenotype and genotype of primary tumors than do serum-cultured cell lines
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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7 April 2017
Stat3 as an oncogene
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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7 April 2017
A randomized trial of bevacizumab for newly diagnosed glioblastoma
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PubMed Central
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29 September 2017
PI3K and STAT3: a new alliance.
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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29 September 2017
Nonreceptor tyrosine kinase BMX maintains self-renewal and tumorigenic potential of glioblastoma stem cells by activating STAT3.
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PubMed Central
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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29 September 2017
Stat3 activation is limiting for reprogramming to ground state pluripotency.
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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29 September 2017
Signal transducer and activator of transcription-3, inflammation, and cancer: how intimate is the relationship?.
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PubMed Central
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29 September 2017
STAT3 is required for proliferation and maintenance of multipotency in glioblastoma stem cells
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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29 September 2017
STAT3 regulation of glioblastoma pathogenesis.
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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29 September 2017
Rembrandt: helping personalized medicine become a reality through integrative translational research
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PubMed Central
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29 September 2017
Activation of nonreceptor tyrosine kinase Bmx/Etk mediated by phosphoinositide 3-kinase, epidermal growth factor receptor, and ErbB3 in prostate cancer cells
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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29 September 2017
Genetic pathways to primary and secondary glioblastoma
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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29 September 2017
STAT3 as a central mediator of neoplastic cellular transformation
1 reference
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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29 September 2017
Tracking STAT nuclear traffic
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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29 September 2017
Inhibition of constitutively active Stat3 suppresses proliferation and induces apoptosis in glioblastoma multiforme cells
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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29 September 2017
STAT signaling in the pathogenesis and treatment of cancer
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PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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29 September 2017
Stat3 activation is required for cellular transformation by v-src
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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29 September 2017
Stat3 activation by Src induces specific gene regulation and is required for cell transformation
1 reference
stated in
PubMed Central
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=4256660
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29 September 2017
Regulation of Stat3 nuclear import by importin alpha5 and importin alpha7 via two different functional sequence elements
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSREP07330
retrieved
21 January 2018
Phosphatidylinositol (3,4,5)P3 is essential but not sufficient for protein kinase B (PKB) activation; phosphatidylinositol (3,4)P2 is required for PKB phosphorylation at Ser-473: studies using cells from SH2-containing inositol-5-phosphatase knockou
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSREP07330
retrieved
21 January 2018
Identifiers
DOI
10.1038/SREP07330
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4878236
Dimensions Publication ID
1001899839
0 references
OpenCitations bibliographic resource ID
4878236
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4878236
PMCID
4256660
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4878236
PubMed ID
25476455
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4878236
ResearchGate publication ID
269187164
0 references
Springer Nature article ID
10.1038/srep07330
0 references
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