(Q28138890)

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Amino-terminal phosphorylation of c-Jun regulates stress-induced apoptosis and cellular proliferation

scientific article (publication date: March 1999)

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Amino-terminal phosphorylation of c-Jun regulates stress-induced apoptosis and cellular proliferation (English)

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A Behrens

1

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Maria Sibilia

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M Sibilia

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2

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E F Wagner

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3

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language of work or name

English

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publication date

March 1999

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published in

Nature Genetics

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volume

21

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issue

3

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page(s)

326-9

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c-Jun is essential for normal mouse development and hepatogenesis

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A null mutation at the c-jun locus causes embryonic lethality and retarded cell growth in culture

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JNK1: a protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain

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The stress-activated protein kinase subfamily of c-Jun kinases

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SEK1 deficiency reveals mitogen-activated protein kinase cascade crossregulation and leads to abnormal hepatogenesis

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The level of intracellular glutathione is a key regulator for the induction of stress-activated signal transduction pathways including Jun N-terminal protein kinases and p38 kinase by alkylating agents

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Induction of c-fos expression through JNK-mediated TCF/Elk-1 phosphorylation.

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The jun proto-oncogene is positively autoregulated by its product, Jun/AP-1

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Intramolecular signal transduction in c-Jun

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Glutamate, but not dopamine, stimulates stress-activated protein kinase and AP-1-mediated transcription in striatal neurons.

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The mechanism of kainic acid neurotoxicity

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7 January 2021

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Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene

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Limbic seizure and brain damage produced by kainic acid: mechanisms and relevance to human temporal lobe epilepsy

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Loss of the p53 tumor suppressor gene protects neurons from kainate-induced cell death.

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Lasting N-terminal phosphorylation of c-Jun and activation of c-Jun N-terminal kinases after neuronal injury.

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Bypass of lethality with mosaic mice generated by Cre– loxP -mediated recombination

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7 January 2021

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The absence of c-fos prevents light-induced apoptotic cell death of photoreceptors in retinal degeneration in vivo

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Strain-dependent epithelial defects in mice lacking the EGF receptor

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Site-specific recombination of a transgene in fertilized eggs by transient expression of Cre recombinase.

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Bax involvement in p53-mediated neuronal cell death

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(Q28138890)

Amino-terminal phosphorylation of c-Jun regulates stress-induced apoptosis and cellular proliferation

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