(Q28145665)

11

object named as

J Tschopp

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Ralph C Budd

2

object named as

R C Budd

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author name string

T Kataoka

1

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N Holler

3

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M Thome

4

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F Martinon

5

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K Burns

7

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M Hahne

8

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N Kennedy

9

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M Kovacsovics

Cell death induction by TNF: a matter of self control

10

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language of work or name

English

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publication date

1 June 2000

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published in

Current Biology

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volume

10

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page(s)

640-8

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issue

11

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cites work

1 reference

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7 January 2021

Death receptors: signaling and modulation

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7 January 2021

Apoptosis by death factor

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7 January 2021

Apoptosis signaling by death receptors.

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7 January 2021

Viral FLICE-inhibitory proteins (FLIPs) prevent apoptosis induced by death receptors

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7 January 2021

Death effector domain-containing herpesvirus and poxvirus proteins inhibit both Fas- and TNFR1-induced apoptosis

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7 January 2021

Inhibition of fas death signals by FLIPs

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7 January 2021

Placing death under control

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7 January 2021

A dominant interfering mutant of FADD/MORT1 enhances deletion of autoreactive thymocytes and inhibits proliferation of mature T lymphocytes

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7 January 2021

A role for FADD in T cell activation and development

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7 January 2021

Fas-mediated apoptosis and activation-induced T-cell proliferation are defective in mice lacking FADD/Mort1

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7 January 2021

FADD: essential for embryo development and signaling from some, but not all, inducers of apoptosis

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7 January 2021

T cell receptor-induced Fas ligand expression in cytotoxic T lymphocyte clones is blocked by protein tyrosine kinase inhibitors and cyclosporin A

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7 January 2021

Fas transduces activation signals in normal human T lymphocytes

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7 January 2021

Expansion or elimination of B cells in vivo: dual roles for CD40- and Fas (CD95)-ligands modulated by the B cell antigen receptor.

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Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas.

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Conversion of membrane-bound Fas(CD95) ligand to its soluble form is associated with downregulation of its proapoptotic activity and loss of liver toxicity

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7 January 2021

AP-1 transcriptional activity requires both T-cell receptor-mediated and co-stimulatory signals in primary T lymphocytes.

1 reference

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7 January 2021

Transcription mediated by NFAT is highly inducible in effector CD4+ T helper 2 (Th2) cells but not in Th1 cells

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7 January 2021

Inhibition of death receptor signals by cellular FLIP

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7 January 2021

The role of c-FLIP in modulation of CD95-induced apoptosis

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7 January 2021

IKAP is a scaffold protein of the IkappaB kinase complex

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7 January 2021

IKK-1 and IKK-2: cytokine-activated IkappaB kinases essential for NF-kappaB activation

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7 January 2021

IRAK: a kinase associated with the interleukin-1 receptor

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7 January 2021

The death domain kinase RIP mediates the TNF-induced NF-kappaB signal

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7 January 2021

Casper is a FADD- and caspase-related inducer of apoptosis

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7 January 2021

Degranulation plays an essential part in regulating cell surface expression of Fas ligand in T cells and natural killer cells

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7 January 2021

Caspase activation is required for T cell proliferation

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7 January 2021

Elevated production of interferon-gamma and interleukin 4 by mature T cells from autoimmune lpr mice correlates with Pgp-1 (CD44) expression.

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7 January 2021

Fas antigen signals proliferation of normal human diploid fibroblast and its mechanism is different from tumor necrosis factor receptor

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7 January 2021

Immune escape of tumors in vivo by expression of cellular FLICE-inhibitory protein

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7 January 2021

Targeted disruption of the mouse Caspase 8 gene ablates cell death induction by the TNF receptors, Fas/Apo1, and DR3 and is lethal prenatally.

1 reference

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7 January 2021

Cell death attenuation by 'Usurpin', a mammalian DED-caspase homologue that precludes caspase-8 recruitment and activation by the CD-95 (Fas, APO-1) receptor complex.

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7 January 2021

MyD88, an adapter protein involved in interleukin-1 signaling

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7 January 2021

Minimal Ras-binding domain of Raf1 can be used as an activation-specific probe for Ras.

1 reference

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7 January 2021

10.1016/S0960-9822(00)00512-1

Identifiers

DOI

1 reference

Consolidated OpenCitations Corpus – April 2017

769619

769619

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

1 reference