(Q28195607)

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Improved endothelial function by the thromboxane A2 receptor antagonist S 18886 in patients with coronary artery disease treated with aspirin

scientific article (publication date: 2 April 2003)

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Improved endothelial function by the thromboxane A2 receptor antagonist S 18886 in patients with coronary artery disease treated with aspirin (English)

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coronary artery disease

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Jean-Luc Dubois-Randé

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Jean-Luc Dubois-Rande

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Serge Adnot

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author name string

Laurent Belhassen

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Gabriel Pelle

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language of work or name

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publication date

2 April 2003

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Journal of the American College of Cardiology

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41

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7

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1198-204

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Evidence of endothelial dysfunction in angiographically normal coronary arteries of patients with coronary artery disease

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Systemic nature of endothelial dysfunction in atherosclerosis

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Endothelium-derived contracting factors

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Glomerular actions of a free radical-generated novel prostaglandin, 8-epi-prostaglandin F2 alpha, in the rat. Evidence for interaction with thromboxane A2 receptors

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A series of prostaglandin F2-like compounds are produced in vivo in humans by a non-cyclooxygenase, free radical-catalyzed mechanism.

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In vivo formation of 8-Epi-prostaglandin F2 alpha is increased in hypercholesterolemia.

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Increased formation of distinct F2 isoprostanes in hypercholesterolemia.

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In vivo formation of 8-iso-prostaglandin f2alpha and platelet activation in diabetes mellitus: effects of improved metabolic control and vitamin E supplementation.

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Oxidant stress and aspirin-insensitive thromboxane biosynthesis in severe unstable angina

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Synthesis and biological evaluation of new tetrahydronaphthalene derivatives as thromboxane receptor antagonists

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Noninvasive determination of endothelium-mediated vasodilation as a screening test for coronary artery disease: pilot study to assess the predictive value in comparison with angina pectoris, exercise electrocardiography, and myocardial perfusion ima

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Molsidomine improves flow-dependent vasodilation in brachial arteries of patients with coronary artery disease

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Endothelial dysfunction in patients with sickle cell disease is related to selective impairment of shear stress-mediated vasodilation

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Assessment of forearm vasodilator responses to acetylcholine and albuterol by strain gauge plethysmography: reproducibility and influence of strain gauge placement

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Aspirin improves endothelial dysfunction in atherosclerosis

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Impairment of forearm vasodilatation to acetylcholine in hypercholesterolemia is reversed by aspirin

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Endothelium-dependent contractions to acetylcholine in the aorta of the spontaneously hypertensive rat

1 reference

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Vasodilation to acetylcholine in primary and secondary forms of human hypertension

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Impaired endothelium-mediated vasodilation in the peripheral vasculature of patients with congestive heart failure

1 reference

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Impaired acetylcholine-mediated vasodilation in patients with congestive heart failure. Role of endothelium-derived vasodilating and vasoconstricting factors

1 reference

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Endothelium-dependent contractions to calcium ionophore A23187, arachidonic acid, and acetylcholine in canine basilar arteries

1 reference

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Oscillatory and steady laminar shear stress differentially affect human endothelial redox state: role of a superoxide-producing NADH oxidase

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Platelet-active drugs : the relationships among dose, effectiveness, and side effects

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Transcellular activation of platelets and endothelial cells by bioactive lipids in platelet microparticles

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The thromboxane receptor antagonist S18886 but not aspirin inhibits atherogenesis in apo E-deficient mice: evidence that eicosanoids other than thromboxane contribute to atherosclerosis

1 reference

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Aspirin-resistant thromboxane biosynthesis and the risk of myocardial infarction, stroke, or cardiovascular death in patients at high risk for cardiovascular events

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