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The APC gene in colorectal cancer
scientific article (publication date: May 2002)
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instance of
scholarly article
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review article
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stated in
Europe PubMed Central
title
The APC gene in colorectal cancer
(English)
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main subject
colorectal cancer
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based on heuristic
inferred from title
colorectal carcinoma
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inferred from title
author
Riccardo Fodde
object named as
R Fodde
series ordinal
1
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language of work or name
English
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publication date
May 2002
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published in
European Journal of Cancer
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volume
38
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issue
7
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page(s)
867-71
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cites work
Cancer statistics, 2000.
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Estimates of the worldwide mortality from 25 cancers in 1990.
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Identification and characterization of the familial adenomatous polyposis coli gene
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Identification of FAP locus genes from chromosome 5q21
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APC mutations occur early during colorectal tumorigenesis
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Mutations of the APC (adenomatous polyposis coli) gene
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Association of the APC tumor suppressor protein with catenins
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Association of the APC gene product with beta-catenin
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Downregulation of beta-catenin by human Axin and its association with the APC tumor suppressor, beta-catenin and GSK3 beta
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Functional interaction of an axin homolog, conductin, with beta-catenin, APC, and GSK3beta
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Domains of axin involved in protein-protein interactions, Wnt pathway inhibition, and intracellular localization
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Axin prevents Wnt-3a-induced accumulation of beta-catenin
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A new member of the frizzled family from Drosophila functions as a Wingless receptor
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Functional interaction of beta-catenin with the transcription factor LEF-1
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XTcf-3 transcription factor mediates beta-catenin-induced axis formation in Xenopus embryos
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Differential molecular interactions of beta-catenin and plakoglobin in adhesion, signaling and cancer
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Constitutive transcriptional activation by a beta-catenin-Tcf complex in APC-/- colon carcinoma
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Activation of beta-catenin-Tcf signaling in colon cancer by mutations in beta-catenin or APC
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Apc1638T: a mouse model delineating critical domains of the adenomatous polyposis coli protein involved in tumorigenesis and development
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Mutations in AXIN2 cause colorectal cancer with defective mismatch repair by activating beta-catenin/TCF signalling
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AXIN1 mutations in hepatocellular carcinomas, and growth suppression in cancer cells by virus-mediated transfer of AXIN1
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Axin and hepatocellular carcinomas.
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Identification of c-MYC as a target of the APC pathway
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Beta-catenin regulates expression of cyclin D1 in colon carcinoma cells
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The cyclin D1 gene is a target of the beta-catenin/LEF-1 pathway
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Immunohistochemical study of epithelial cell proliferation in hyperplastic polyps, adenomas, and adenocarcinomas of the large bowel
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Proliferation and apoptosis in proliferative lesions of the colon and rectum
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Immunohistochemical analysis of cell kinetic parameters in colonic adenocarcinomas, adenomas, and normal mucosa
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The metalloproteinase matrilysin is a target of beta-catenin transactivation in intestinal tumors
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beta-catenin regulates the expression of the matrix metalloproteinase-7 in human colorectal cancer
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Expression of CD44 in Apc and Tcf mutant mice implies regulation by the WNT pathway
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Expression of nuclear beta-catenin and c-myc is correlated with tumor size but not with proliferative activity of colorectal adenomas
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Target genes of beta-catenin-T cell-factor/lymphoid-enhancer-factor signaling in human colorectal carcinomas
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The APC gene product in normal and tumor cells
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APC expression in normal human tissues
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Depletion of epithelial stem-cell compartments in the small intestine of mice lacking Tcf-4
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Restricted high level expression of Tcf-4 protein in intestinal and mammary gland epithelium
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7 January 2021
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Oncogene expression in patients with familial polyposis coli/Gardner's syndrome
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Synergy between tumor suppressor APC and the beta-catenin-Tcf4 target Tcf1.
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Reciprocity between membranous and nuclear expression of beta-catenin in colorectal tumours
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Nuclear overexpression of the oncoprotein beta-catenin in colorectal cancer is localized predominantly at the invasion front.
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E-cadherin and adenomatous polyposis coli mutations are synergistic in intestinal tumor initiation in mice
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E-Cadherin Suppresses Cellular Transformation by Inhibiting β-Catenin Signaling in an Adhesion-Independent Manner
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Mutations in the APC tumour suppressor gene cause chromosomal instability
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A role for the Adenomatous Polyposis Coli protein in chromosome segregation
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The centrosome--a tiny organelle with big potential
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Mechanisms underlying losses of heterozygosity in human colorectal cancers
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Abnormal centrosome amplification in the absence of p53.
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Genomic instability and apoptosis are frequent in p53 deficient young mice
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Defects in transforming growth factor-beta signaling cooperate with a Ras oncogene to cause rapid aneuploidy and malignant transformation of mouse keratinocytes
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C-myc overexpression and p53 loss cooperate to promote genomic instability
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c-Myc overexpression uncouples DNA replication from mitosis
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Chromosome instability contributes to loss of heterozygosity in mice lacking p53
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APC, signal transduction and genetic instability in colorectal cancer
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Identifiers
DOI
10.1016/S0959-8049(02)00040-0
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1502172
OpenCitations bibliographic resource ID
1502172
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1502172
PubMed publication ID
11978510
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1502172
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