(Q28267760)

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CELL BIOLOGY. GATA get a hold on senescence

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CELL BIOLOGY. GATA get a hold on senescence (English)

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Liam D Cassidy

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Masashi Narita

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Masashi Narita

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2

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language of work or name

English

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publication date

25 September 2015

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published in

Science

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volume

349

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issue

6255

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page(s)

1448-9

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cites work

Autophagy mediates the mitotic senescence transition

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The senescence-associated secretory phenotype: the dark side of tumor suppression

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Inside and out: the activities of senescence in cancer.

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The DNA damage response induces inflammation and senescence by inhibiting autophagy of GATA4.

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Interactions between autophagy receptors and ubiquitin-like proteins form the molecular basis for selective autophagy.

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p38MAPK is a novel DNA damage response-independent regulator of the senescence-associated secretory phenotype

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IKKi: a novel regulator of Act1, IL-17 signaling and pulmonary inflammation

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p62: a versatile multitasker takes on cancer

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Assurance of mitochondrial integrity and mammalian longevity by the p62-Keap1-Nrf2-Nqo1 cascade

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Spatial coupling of mTOR and autophagy augments secretory phenotypes.

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MTOR regulates the pro-tumorigenic senescence-associated secretory phenotype by promoting IL1A translation

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DOI

10.1126/SCIENCE.AAD2501

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PubMed publication ID

26404812

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(Q28267760)

CELL BIOLOGY. GATA get a hold on senescence

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