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RNA editing of brain glutamate receptor channels: mechanism and physiology
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scholarly article
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Europe PubMed Central
title
RNA editing of brain glutamate receptor channels: mechanism and physiology
(English)
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main subject
brain
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physiology
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author
Rolf Sprengel
series ordinal
3
object named as
R Sprengel
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Peter H. Seeburg
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1
object named as
P H Seeburg
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author name string
M Higuchi
series ordinal
2
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language of work or name
English
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publication date
May 1998
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published in
Brain Research Reviews
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volume
26
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page(s)
217-29
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issue
2-3
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cites work
A novel form of tissue-specific RNA processing produces apolipoprotein-B48 in intestine
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Apolipoprotein B RNA editing enzyme-deficient mice are viable despite alterations in lipoprotein metabolism
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RNA editing. An I for editing
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RNA editing in brain controls a determinant of ion flow in glutamate-gated channels
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The physiology of excitatory amino acids in the vertebrate central nervous system
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RNA editing
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RNA editing of AMPA receptor subunit GluR-B: a base-paired intron-exon structure determines position and efficiency
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A place in the world for RNA editing
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An unwinding activity that covalently modifies its double-stranded RNA substrate
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Double-stranded RNA unwinding and modifying activity is detected ubiquitously in primary tissues and cell lines
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The role of glutamate neurotoxicity in hypoxic-ischemic neuronal death
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Early-onset epilepsy and postnatal lethality associated with an editing-deficient GluR-B allele in mice
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Molecular mechanisms controlling calcium entry through AMPA-type glutamate receptor channels
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Cloned glutamate receptors
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The rat delta-1 and delta-2 subunits extend the excitatory amino acid receptor family
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Neurodegeneration in Lurcher mice caused by mutation in delta2 glutamate receptor gene.
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Impairment of motor coordination, Purkinje cell synapse formation, and cerebellar long-term depression in GluR delta 2 mutant mice
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Identifiers
DOI
10.1016/S0165-0173(97)00062-3
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4260269
OpenCitations bibliographic resource ID
4260269
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4260269
PubMed ID
9651532
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4260269
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