(Q28278230)

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Proteomic analysis of vascular endothelial growth factor-induced endothelial cell differentiation reveals a role for chloride intracellular channel 4 (CLIC4) in tubular morphogenesis

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Proteomic analysis of vascular endothelial growth factor-induced endothelial cell differentiation reveals a role for chloride intracellular channel 4 (CLIC4) in tubular morphogenesis (English)

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based on heuristic

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cell differentiation

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based on heuristic

inferred from title

6

object named as

Stuart Yuspa

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Lena Claesson-Welsh

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object named as

Lena Claesson-Welsh

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author name string

Svante Bohman

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Taro Matsumoto

2

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Kwang Suh

3

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Anna Dimberg

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Lars Jakobsson

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language of work or name

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publication date

23 December 2005

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Journal of Biological Chemistry

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280

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51

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42397-404

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Mechanisms of angiogenesis

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Angiogenesis-dependent diseases

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Endogenous angiogenesis inhibitors

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Abnormal blood vessel development and lethality in embryos lacking a single VEGF allele

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Heterozygous embryonic lethality induced by targeted inactivation of the VEGF gene

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VEGF guides angiogenic sprouting utilizing endothelial tip cell filopodia

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Postnatal vasculogenesis

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Angiogenesis assays: a critical overview

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In vitro rapid organization of endothelial cells into capillary-like networks is promoted by collagen matrices

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Tissue engineering: creation of long-lasting blood vessels

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Gene expression profiling in an in vitro model of angiogenesis

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p53 and tumor necrosis factor alpha regulate the expression of a mitochondrial chloride channel protein

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M506724200

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Overexpressed chloride intracellular channel protein CLIC4 (p64H1) is an essential component of novel plasma membrane anion channels

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Chloride intracellular channel protein CLIC4 (p64H1) binds directly to brain dynamin I in a complex containing actin, tubulin and 14-3-3 isoforms

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mtCLIC/CLIC4, an organellular chloride channel protein, is increased by DNA damage and participates in the apoptotic response to p53

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Differential expression of a chloride intracellular channel gene, CLIC4, in transforming growth factor-beta1-mediated conversion of fibroblasts to myofibroblasts

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A C. elegans CLIC-like protein required for intracellular tube formation and maintenance.

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Induction of tubulogenesis in telomerase-immortalized human microvascular endothelial cells by glioblastoma cells

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Mass Spectrometric Sequencing of Proteins from Silver-Stained Polyacrylamide Gels

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Deregulation of Flk-1/vascular endothelial growth factor receptor-2 in fibroblast growth factor receptor-1-deficient vascular stem cell development.

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Phosphoinositide 3 kinase is critical for survival, mitogenesis and migration but not for differentiation of endothelial cells

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p38 MAP kinase negatively regulates endothelial cell survival, proliferation, and differentiation in FGF-2-stimulated angiogenesis

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p38 MAP kinase activation by vascular endothelial growth factor mediates actin reorganization and cell migration in human endothelial cells

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Subtractive proteomic mapping of the endothelial surface in lung and solid tumours for tissue-specific therapy

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Fibroblast growth factor receptor-1 expression is required for hematopoietic but not endothelial cell development

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Cellular abnormalities of blood vessels as targets in cancer

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Rat brain p64H1, expression of a new member of the p64 chloride channel protein family in endoplasmic reticulum

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Angiostatin binds to tyrosine kinase substrate annexin II through the lysine-binding domain in endothelial cells

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A comparison of selected mRNA and protein abundances in human liver

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Modulation of gene expression by hypoxia in human umbilical cord vein endothelial cells: A transcriptomic and proteomic study

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M506724200

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CLIC1 inserts from the aqueous phase into phospholipid membranes, where it functions as an anion channel

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M506724200

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Angiogenesis in vitro

1 reference

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21 January 2018

Identifiers

10.1074/JBC.M506724200

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

OpenCitations bibliographic resource ID

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

PubMed publication ID

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

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