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Cell death of dopamine neurons in aging and Parkinson's disease
scientific article
Cell death of dopamine neurons in aging and Parkinson’s disease
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scholarly article
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title
Cell death of dopamine neurons in aging and Parkinson's disease
(English)
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main subject
Parkinson's disease
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inferred from title
dopamine
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cell death
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inferred from title
author name string
M Naoi
series ordinal
1
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W Maruyama
series ordinal
2
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language of work or name
English
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publication date
November 1999
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published in
Mechanisms of Ageing and Development
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volume
111
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issue
2-3
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page(s)
175-88
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cites work
Mitochondrial DNA deletions in human brain: regional variability and increase with advanced age
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Oxidative inactivation of tyrosine hydroxylase in substantia nigra of aged rat
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Ageing and Parkinson's disease: substantia nigra regional selectivity
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Oxidative stress and the pathogenesis of Parkinson's disease
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Aging produces a specific pattern of striatal dopamine loss: implications for the etiology of idiopathic Parkinson's disease
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Mutations in the parkin gene cause autosomal recessive juvenile parkinsonism
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Possible role of neuromelanin in the pathogenesis of Parkinson's disease
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Parkinson's disease
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N-methylation of dopamine-derived 6,7-dihydroxy-1,2,3,4-tetrahydroisoquinoline, (R)-salsolinol, in rat brains: in vivo microdialysis study.
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N-methyl(R)salsolinol produces hydroxyl radicals: involvement to neurotoxicity
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Dopamine-derived 1-methyl-6,7-dihydroxyisoquinolines as hydroxyl radical promoters and scavengers in the rat brain: in vivo and in vitro studies
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A dopaminergic neurotoxin, (R)-N-methylsalsolinol, increases in parkinsonian cerebrospinal fluid
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Stereospecific occurrence of a parkinsonism-inducing catechol isoquinoline, N-methyl(R)salsolinol, in the human intraventricular fluid
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A dopaminergic neurotoxin, 1(R), 2(N)-dimethyl-6,7-dihydroxy-1,2,3,4-tetrahydroisoquinoline, N-methyl(R)salsolinol, and its oxidation product, 1,2(N)-dimethyl-6,7-dihydroxyisoquinolinium ion, accumulate in the nigro-striatal system of the human brai
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An endogenous dopaminergic neurotoxin, N-methyl-(R)-salsolinol, induces DNA damage in human dopaminergic neuroblastoma SH-SY5Y cells
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A neurotoxin N-methyl(R)salsolinol induces apoptotic cell death in differentiated human dopaminergic neuroblastoma SH-SY5Y cells
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Rate of cell death in parkinsonism indicates active neuropathological process
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Histochemical detection of apoptosis in Parkinson's disease.
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Effects of various tetrahydroisoquinoline derivatives on mitochondrial respiration and the electron transfer complexes
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Binding of 1,2(N)-dimethyl-6,7-dihydroxy-isoquinolinium ion to melanin: effects of ferrous and ferric ion on the binding
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Enzymatic oxidation of the dopaminergic neurotoxin, 1(R), 2(N)-dimethyl-6,7-dihydroxy-1,2,3,4-tetrahydroisoquinoline, into 1,2(N)-dimethyl-6,7-dihydroxyisoquinolinium ion
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Dopamine-derived endogenous 1(R),2(N)-dimethyl-6,7-dihydroxy- 1,2,3,4-tetrahydroisoquinoline, N-methyl-(R)-salsolinol, induced parkinsonism in rat: biochemical, pathological and behavioral studies.
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A novel enzyme enantio-selectively synthesizes (R)salsolinol, a precursor of a dopaminergic neurotoxin, N-methyl(R)salsolinol.
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A neutral N-methyltransferase activity in the striatum determines the level of an endogenous MPP+-like neurotoxin, 1,2-dimethyl-6,7-dihydroxyisoquinolinium ion, in the substantia nigra of human brains.
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N-methyl-(R)salsolinol as a dopaminergic neurotoxin: from an animal model to an early marker of Parkinson's disease.
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(R)salsolinol N-methyltransferase activity increases in parkinsonian lymphocytes
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Microelectrophoretic study of radiation-induced DNA damages in individual mammalian cells
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Mutation in the alpha-synuclein gene identified in families with Parkinson's disease
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Formation of 6-hydroxydopamine in caudate nucleus of the rat brain after a single large dose of methylamphetamine.
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inferred from DOI database lookup
Cytotoxicity of endogenous isoquinolines to human dopaminergic neuroblastoma SH-SY5Y cells.
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0047-6374%2899%2900064-0
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Alkaloids in mammalian tissues. 2. Synthesis of (+)- and (-)-1-substituted-6,7-dihydroxy-1,2,3,4-tetrahydroisoquinolines
1 reference
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Crossref
reference URL
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Identifiers
DOI
10.1016/S0047-6374(99)00064-0
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
2472973
OpenCitations bibliographic resource ID
2472973
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
2472973
PubMed ID
10656535
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
2472973
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